Felix Mulenge, Olivia Luise Gern, Lena Mareike Busker, Angela Aringo, Luca Ghita, Inken Waltl, Andreas Pavlou, Ulrich Kalinke
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引用次数: 0
摘要
小胶质细胞是抵御神经侵袭性病毒感染的前线防线,然而,确定小胶质细胞在健康和疾病条件下的实际转录情况却具有挑战性。在这里,我们采用了多种实验方法来描述病毒感染期间小胶质细胞的转录情况。有趣的是,我们发现在分选的小胶质细胞中,多个激活基因被人为诱导,而且我们证明细胞分选过程中遇到的剪切应力是关键诱导因素之一。事后分析表明,公开的大规模单细胞 RNA 测序数据集明显受到与细胞分选相关的异常特征的影响。通过利用核糖体标记方法,我们开发了一种策略,通过比较免疫沉淀 RNA 和总 RNA 来富集小胶质细胞特异性转录本。这些富集的转录本有助于确定小胶质细胞在健康和病毒感染条件下的真正特征。这些统一的小胶质细胞特征可作为一个基准,用于回顾性评估现有图谱中的体外伪影。利用小胶质细胞转译组,我们发现在 VSV 感染过程中,与 T 细胞活化和细胞因子产生有关的基因得到了丰富。这些数据将小胶质细胞与 T 细胞再刺激联系起来,进一步强调了小胶质细胞参与了大脑中抗病毒 T 细胞反应的形成。总之,我们的研究界定了小胶质细胞在稳态和病毒性脑炎期间的转录情况,并强调了小胶质细胞和 T 细胞之间有助于控制病毒传播的细胞相互作用。
Transcriptomic analysis unveils bona fide molecular signatures of microglia under conditions of homeostasis and viral encephalitis.
Microglia serve as a front-line defense against neuroinvasive viral infection, however, determination of their actual transcriptional profiles under conditions of health and disease is challenging. Here, we used various experimental approaches to delineate the transcriptional landscape of microglia during viral infection. Intriguingly, multiple activation genes were found to be artificially induced in sorted microglia and we demonstrated that shear stress encountered during cell sorting was one of the key inducers. Post-hoc analysis revealed that publicly available large-scale single-cell RNA sequencing datasets were significantly tainted by aberrant signatures that are associated with cell sorting. By exploiting the ribosomal tagging approach, we developed a strategy to enrich microglia-specific transcripts by comparing immunoprecipitated RNA with total RNA. Such enriched transcripts were instrumental in defining bona fide signatures of microglia under conditions of health and virus infection. These unified microglial signatures may serve as a benchmark to retrospectively assess ex vivo artefacts from available atlases. Leveraging the microglial translatome, we found enrichment of genes implicated in T-cell activation and cytokine production during the course of VSV infection. These data linked microglia with T-cell re-stimulation and further underscored that microglia are involved in shaping antiviral T-cell responses in the brain. Collectively, our study defines the transcriptional landscape of microglia under steady state and during viral encephalitis and highlights cellular interactions between microglia and T cells that contribute to the control of virus dissemination.
期刊介绍:
The Journal of Neuroinflammation is a peer-reviewed, open access publication that emphasizes the interaction between the immune system, particularly the innate immune system, and the nervous system. It covers various aspects, including the involvement of CNS immune mediators like microglia and astrocytes, the cytokines and chemokines they produce, and the influence of peripheral neuro-immune interactions, T cells, monocytes, complement proteins, acute phase proteins, oxidative injury, and related molecular processes.
Neuroinflammation is a rapidly expanding field that has significantly enhanced our knowledge of chronic neurological diseases. It attracts researchers from diverse disciplines such as pathology, biochemistry, molecular biology, genetics, clinical medicine, and epidemiology. Substantial contributions to this field have been made through studies involving populations, patients, postmortem tissues, animal models, and in vitro systems.
The Journal of Neuroinflammation consolidates research that centers around common pathogenic processes. It serves as a platform for integrative reviews and commentaries in this field.