[撤稿】SFRP2 通过 Wnt 通路调控线粒体分裂,从而调节非小细胞肺癌 A549 细胞的凋亡和转移。

IF 3.4 3区 医学 Q2 MEDICINE, RESEARCH & EXPERIMENTAL Molecular medicine reports Pub Date : 2024-10-01 Epub Date: 2024-08-19 DOI:10.3892/mmr.2024.13305
Peng Li, Shu Zhao, Yi Hu
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引用次数: 0

摘要

在这篇论文发表后,一位关心此事的读者提请编辑注意,第 1928 页图 2C 中显示的某些 JC1 染色的细胞图像与不同研究机构不同作者撰写的另一篇文章中以不同形式发表的数据惊人地相似[Yao S and Yan W., 2009]:过表达 Mst1 通过抑制 AMPK-Sirt3 通路和激活线粒体裂变降低胃癌细胞活力。Onco Targets Ther 11: 8465-8479, 2019]。由于上述文章中存在争议的数据在提交给《分子医学报告》之前已经发表,编辑决定从《分子医学报告》上撤回这篇论文。编辑部要求作者对这些问题做出解释,但没有得到满意的答复。对于给读者带来的不便,编辑深表歉意。[分子医学报告 20: 1925-1932, 2019; DOI: 10.3892/mmr.2019.10393]。
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[Retracted] SFRP2 modulates non‑small cell lung cancer A549 cell apoptosis and metastasis by regulating mitochondrial fission via Wnt pathways.

Following the publication of this paper, it was drawn to the Editor's attention by a concerned reader that certain of the JC1‑stained cellular images shown in Fig. 2C on p. 1928 were strikingly similar to data that had already been published in different form in another article written by different authors at different research institutes [Yao S and Yan W: Overexpression of Mst1 reduces gastric cancer cell viability by repressing the AMPK‑Sirt3 pathway and activating mitochondrial fission. Onco Targets Ther 11: 8465‑8479, 2019]. Owing to the fact that the contentious data in the above article had already been published prior to its submission to Molecular Medicine Reports, the Editor has decided that this paper should be retracted from the Journal. The authors were asked for an explanation to account for these concerns, but the Editorial Office did not receive a satisfactory reply. The Editor apologizes to the readership for any inconvenience caused. [Molecular Medicine Reports 20: 1925‑1932, 2019; DOI: 10.3892/mmr.2019.10393].

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来源期刊
Molecular medicine reports
Molecular medicine reports 医学-病理学
CiteScore
7.60
自引率
0.00%
发文量
321
审稿时长
1.5 months
期刊介绍: Molecular Medicine Reports is a monthly, peer-reviewed journal available in print and online, that includes studies devoted to molecular medicine, underscoring aspects including pharmacology, pathology, genetics, neurosciences, infectious diseases, molecular cardiology and molecular surgery. In vitro and in vivo studies of experimental model systems pertaining to the mechanisms of a variety of diseases offer researchers the necessary tools and knowledge with which to aid the diagnosis and treatment of human diseases.
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