Ca2+依赖性脂质偏好决定突触表敏-1 C2A和C2B的动态:实验和模拟的启示

IF 4.4 2区 生物学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY Structure Pub Date : 2024-08-21 DOI:10.1016/j.str.2024.07.017
Julian Bender, Til Kundlacz, Lucas S.P. Rudden, Melissa Frick, Julia Bieber, Matteo T. Degiacomi, Carla Schmidt
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引用次数: 0

摘要

神经元之间的信号传递需要神经递质从突触小泡的内腔外排到突触间隙。在 Ca2+ 流入后,Ca2+ 传感器 synaptotagmin-1 会促进这一过程。其基本机制涉及静电和疏水相互作用,调整突触塔格明-1 的两个 C2 结构域的脂质偏好;然而,有关细节的讨论仍存在争议。因此,我们采用了一种多学科方法,对分离的 C2A 和 C2B 结构域的脂质和膜结合进行了表征。我们首先以与单个脂质物种的相互作用为目标,然后研究与脂质体模型膜的相互作用。最后,我们进行了分子动力学模拟,以揭示膜结合的差异。我们发现,作为对 Ca2+ 的反应,两个 C2 结构域都会插入脂膜;但是,C2A 采用了更加垂直的取向,而 C2B 则保持平行。这些发现使我们能够提出突触柄蛋白-1在膜融合过程中的机制。
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Ca2+-dependent lipid preferences shape synaptotagmin-1 C2A and C2B dynamics: Insights from experiments and simulations

Signal transmission between neurons requires exocytosis of neurotransmitters from the lumen of synaptic vesicles into the synaptic cleft. Following an influx of Ca2+, this process is facilitated by the Ca2+ sensor synaptotagmin-1. The underlying mechanisms involve electrostatic and hydrophobic interactions tuning the lipid preferences of the two C2 domains of synaptotagmin-1; however, the details are still controversially discussed. We, therefore, follow a multidisciplinary approach and characterize lipid and membrane binding of the isolated C2A and C2B domains. We first target interactions with individual lipid species, and then study interactions with model membranes of liposomes. Finally, we perform molecular dynamics simulations to unravel differences in membrane binding. We found that both C2 domains, as a response to Ca2+, insert into the lipid membrane; however, C2A adopts a more perpendicular orientation while C2B remains parallel. These findings allow us to propose a mechanism for synaptotagmin-1 during membrane fusion.

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来源期刊
Structure
Structure 生物-生化与分子生物学
CiteScore
8.90
自引率
1.80%
发文量
155
审稿时长
3-8 weeks
期刊介绍: Structure aims to publish papers of exceptional interest in the field of structural biology. The journal strives to be essential reading for structural biologists, as well as biologists and biochemists that are interested in macromolecular structure and function. Structure strongly encourages the submission of manuscripts that present structural and molecular insights into biological function and mechanism. Other reports that address fundamental questions in structural biology, such as structure-based examinations of protein evolution, folding, and/or design, will also be considered. We will consider the application of any method, experimental or computational, at high or low resolution, to conduct structural investigations, as long as the method is appropriate for the biological, functional, and mechanistic question(s) being addressed. Likewise, reports describing single-molecule analysis of biological mechanisms are welcome. In general, the editors encourage submission of experimental structural studies that are enriched by an analysis of structure-activity relationships and will not consider studies that solely report structural information unless the structure or analysis is of exceptional and broad interest. Studies reporting only homology models, de novo models, or molecular dynamics simulations are also discouraged unless the models are informed by or validated by novel experimental data; rationalization of a large body of existing experimental evidence and making testable predictions based on a model or simulation is often not considered sufficient.
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