扩展由致病性 ARSG 变异引起的 IV 型乌谢尔综合征的遗传图谱。

IF 2.9 3区 医学 Q2 GENETICS & HEREDITY Clinical Genetics Pub Date : 2024-08-28 DOI:10.1111/cge.14614
Miriam Bauwens, Vincent De Man, Isabelle Audo, Irina Balikova, Wadih M Zein, Vasily Smirnov, Sebastian Held, Sascha Vermeer, Elke Loos, Julie Jacob, Ingele Casteels, Julie Désir, Fanny Depasse, Stijn Van de Sompele, Mattias Van Heetvelde, Marieke De Bruyne, Camille Andrieu, Christel Condroyer, Aline Antonio, Robert Hufnagel, Ana Luísa Carvalho, João Pedro Marques, Christina Zeitz, Elfride De Baere, Markus Damme
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引用次数: 0

摘要

乌谢尔综合征(USH)是最常见的聋盲病因。乌谢氏综合征为常染色体隐性遗传,以杆-锥体营养不良或视网膜色素变性(RP)为特征,通常伴有感音神经性听力损失。目前已发现超过 15 个基因的变异可导致临床和遗传学上不同的亚型。这种亚型被归为 "USH IV",RP发病较晚,通常为晚发性进行性SNHL,无前庭受累。在这里,我们描述了九名新受试者和四例 USH IV 表型病例的临床描述,其中有七个新的和两个已知的致病变体。功能实验表明,异位表达突变的 ARSG cDNA 时,硫酸酯酶酶活性完全丧失。有趣的是,我们还发现了一个同卵错义变体 p.(Arg99His) ,该变体以前曾在神经细胞类脂质沉着病犬中被描述过。我们的研究扩大了 ARSG-USH IV 的遗传范围,使已知受试者的数量增加了 30% 以上。这些发现突出表明,USH IV 很可能诊断不足,并强调有必要对未解决的聋盲综合征受试者进行分子检测。最后,在对明显的孤立遗传性视网膜疾病进行遗传学检查时,应考虑检测 ARSG。
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Expanding the genetic landscape of Usher syndrome type IV caused by pathogenic ARSG variants.

Usher syndrome (USH) is the most common cause of deafblindness. USH is autosomal recessively inherited and characterized by rod-cone dystrophy or retinitis pigmentosa (RP), often accompanied by sensorineural hearing loss. Variants in >15 genes have been identified as causative for clinically and genetically distinct subtypes. Among the ultra-rare and recently discovered genes is ARSG, coding for the lysosomal sulfatase Arylsulfatase G. This subtype was assigned as "USH IV" with a late onset of RP and usually late-onset progressive SNHL without vestibular involvement. Here, we describe nine new subjects and the clinical description of four cases with the USH IV phenotype bearing seven novel and two known pathogenic variants. Functional experiments indicated the complete loss of sulfatase enzymatic activity upon ectopic expression of mutated ARSG cDNA. Interestingly, we identified a homozygous missense variant, p.(Arg99His), previously described in dogs with neuronal ceroid lipofuscinosis. Our study expands the genetic landscape of ARSG-USH IV and the number of known subjects by more than 30%. These findings highlight that USH IV likely has been underdiagnosed and emphasize the need to test molecularly unresolved subjects with deafblindness syndrome. Finally, testing of ARSG should be considered for the genetic work-up of apparent isolated inherited retinal diseases.

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来源期刊
Clinical Genetics
Clinical Genetics 医学-遗传学
CiteScore
6.50
自引率
0.00%
发文量
175
审稿时长
3-8 weeks
期刊介绍: Clinical Genetics links research to the clinic, translating advances in our understanding of the molecular basis of genetic disease for the practising clinical geneticist. The journal publishes high quality research papers, short reports, reviews and mini-reviews that connect medical genetics research with clinical practice. Topics of particular interest are: • Linking genetic variations to disease • Genome rearrangements and disease • Epigenetics and disease • The translation of genotype to phenotype • Genetics of complex disease • Management/intervention of genetic diseases • Novel therapies for genetic diseases • Developmental biology, as it relates to clinical genetics • Social science research on the psychological and behavioural aspects of living with or being at risk of genetic disease
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