Jing-jing Mao , Xiao-yu Dai , Yun-zi Liu , Li-jun Ren , Ji-qian-zhu Zhang , Lang Yan , Jin-feng Li , Yi-jun Tian , Jiang-bo Zhu , Ji-kuai Chen
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We observed that DNAJA1 protein levels increased on postnatal days 14, 21, and 28, peaking on postnatal day 35 in mice. Immunofluorescence staining indicated that DNAJA1 expression varies across different stages of the spermatogenesis cycle. Additionally, DNAJA1 was absent in epididymal sperm. In early- and mid-stage tubules, DNAJA1 protein distribution was co-localized with residual bodies in elongating spermatids. Furthermore, we found that DNAJA1 knockout significantly reduced protein polyubiquitination in the testis. Analysis of the GEO database showed that DNAJA1 levels were significantly decreased in semen samples from subjects with teratozoospermia, asthenozoospermia, and impaired spermatogenesis. Our findings suggest that DNAJA1 is an essential protein for spermatogenesis, and its deletion reduces protein polyubiquitination in the testis, ultimately resulting in infertility and spermatogenesis defects.</p></div>","PeriodicalId":21137,"journal":{"name":"Reproductive toxicology","volume":"130 ","pages":"Article 108701"},"PeriodicalIF":3.3000,"publicationDate":"2024-08-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"DNAJA1 regulates protein ubiquitination and is essential for spermatogenesis in the testes of mice and rats\",\"authors\":\"Jing-jing Mao , Xiao-yu Dai , Yun-zi Liu , Li-jun Ren , Ji-qian-zhu Zhang , Lang Yan , Jin-feng Li , Yi-jun Tian , Jiang-bo Zhu , Ji-kuai Chen\",\"doi\":\"10.1016/j.reprotox.2024.108701\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<div><p>DNAJA1 is a member of type I DnaJ proteins, which is essential for spermatogenesis and male fertility. 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Furthermore, we found that DNAJA1 knockout significantly reduced protein polyubiquitination in the testis. Analysis of the GEO database showed that DNAJA1 levels were significantly decreased in semen samples from subjects with teratozoospermia, asthenozoospermia, and impaired spermatogenesis. 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引用次数: 0
摘要
DNAJA1 是 I 型 DnaJ 蛋白的成员,对精子发生和男性生育至关重要。然而,它在睾丸中的表达模式及其对精子发生的影响仍不清楚。我们的研究旨在阐明DNAJA1的作用机制。本研究采用了DNAJA1基因敲除小鼠。我们采用了DNAJA1基因敲除小鼠,并进行了Western印迹和免疫荧光分析,以确定DNAJA1在睾丸不同发育阶段的蛋白丰度。结果显示,DNAJA1主要在睾丸中表达,基因敲除会导致雄性小鼠完全不育。我们观察到,DNAJA1蛋白水平在小鼠出生后第14、21和28天增加,在出生后第35天达到高峰。免疫荧光染色表明,DNAJA1的表达在精子发生周期的不同阶段有所不同。此外,附睾精子中没有DNAJA1。在早期和中期的小管中,DNAJA1蛋白分布与伸长精子中的残留体共同定位。此外,我们还发现 DNAJA1 基因敲除会显著减少睾丸中蛋白质的多泛素化。对GEO数据库的分析表明,在畸形精子症、无精子症和精子发生障碍患者的精液样本中,DNAJA1的水平明显下降。我们的研究结果表明,DNAJA1是精子发生过程中必不可少的蛋白质,其缺失会减少蛋白质在睾丸中的多泛素化,最终导致不育和精子发生缺陷。
DNAJA1 regulates protein ubiquitination and is essential for spermatogenesis in the testes of mice and rats
DNAJA1 is a member of type I DnaJ proteins, which is essential for spermatogenesis and male fertility. However, its expression pattern in the testes and its impact on spermatogenesis remains unclear. Our study aimed to elucidate the mechanism of action of DNAJA1. We employed DNAJA1 knockout mice in this study. Western blotting and immunofluorescence analysis were conducted to determine the protein abundance of DNAJA1 in testes at various developmental stages. Our results revealed that DNAJA1 is predominantly expressed in the testes, and its knockout leads to complete infertility in male mice. We observed that DNAJA1 protein levels increased on postnatal days 14, 21, and 28, peaking on postnatal day 35 in mice. Immunofluorescence staining indicated that DNAJA1 expression varies across different stages of the spermatogenesis cycle. Additionally, DNAJA1 was absent in epididymal sperm. In early- and mid-stage tubules, DNAJA1 protein distribution was co-localized with residual bodies in elongating spermatids. Furthermore, we found that DNAJA1 knockout significantly reduced protein polyubiquitination in the testis. Analysis of the GEO database showed that DNAJA1 levels were significantly decreased in semen samples from subjects with teratozoospermia, asthenozoospermia, and impaired spermatogenesis. Our findings suggest that DNAJA1 is an essential protein for spermatogenesis, and its deletion reduces protein polyubiquitination in the testis, ultimately resulting in infertility and spermatogenesis defects.
期刊介绍:
Drawing from a large number of disciplines, Reproductive Toxicology publishes timely, original research on the influence of chemical and physical agents on reproduction. Written by and for obstetricians, pediatricians, embryologists, teratologists, geneticists, toxicologists, andrologists, and others interested in detecting potential reproductive hazards, the journal is a forum for communication among researchers and practitioners. Articles focus on the application of in vitro, animal and clinical research to the practice of clinical medicine.
All aspects of reproduction are within the scope of Reproductive Toxicology, including the formation and maturation of male and female gametes, sexual function, the events surrounding the fusion of gametes and the development of the fertilized ovum, nourishment and transport of the conceptus within the genital tract, implantation, embryogenesis, intrauterine growth, placentation and placental function, parturition, lactation and neonatal survival. Adverse reproductive effects in males will be considered as significant as adverse effects occurring in females. To provide a balanced presentation of approaches, equal emphasis will be given to clinical and animal or in vitro work. Typical end points that will be studied by contributors include infertility, sexual dysfunction, spontaneous abortion, malformations, abnormal histogenesis, stillbirth, intrauterine growth retardation, prematurity, behavioral abnormalities, and perinatal mortality.