G 蛋白偶联受体 GPR56 是 NK 细胞迁移的抑制性检查点

IF 3.6 3区 医学 Q2 IMMUNOLOGY Journal of immunology Pub Date : 2024-09-25 DOI:10.4049/jimmunol.2400228
Daniel Palacios, Rakesh Kumar Majhi, Edina K Szabo, Dennis Clement, Mieszko Lachota, Herman Netskar, Leena Penna, Silje Z Krokeide, Marianna Vincenti, Lise Kveberg, Karl-Johan Malmberg
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引用次数: 0

摘要

G 蛋白偶联受体(GPCR)是最大的表面受体家族,负责细胞生长、神经传递、激素释放和细胞迁移等关键生理功能。由 ADGRG1 编码的 GPCR 56 (GPR56) 是一种粘附性 GPCR,存在于多种类型的细胞中,包括神经祖细胞、黑色素瘤细胞和淋巴细胞,如效应记忆 T 细胞、γδ T 细胞和 NK 细胞。利用 RNA 序列分析和高分辨率流式细胞术,我们发现 GPR56 mRNA 和蛋白质的表达随着 NK 细胞的分化而增加,并在适应性 NK 细胞中达到峰值。小干扰 RNA 沉默 GPR56 会导致自发迁移和趋化因子诱导的迁移增加,这表明 GPR56 在高度分化的 NK 细胞迁移过程中起着上游检查点的作用。对 GPR56 的激动刺激也可诱导 NK 细胞迁移的增加,从而导致受体的快速内化和失活。从机理上讲,GPR56 的结扎和下调与具有 PDZ 结合基调的转录辅激活因子转位到细胞核和肌动蛋白聚合增加有关。这些数据共同揭示了 GPR56 在人类 NK 细胞亚群迁移行为中的作用,并为通过释放迁移检查点来改善 NK 细胞对癌症组织的浸润提供了可能性。
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The G Protein-Coupled Receptor GPR56 Is an Inhibitory Checkpoint for NK Cell Migration.

G protein-coupled receptors (GPCRs) represent the largest family of surface receptors and are responsible for key physiological functions, including cell growth, neurotransmission, hormone release, and cell migration. The GPCR 56 (GPR56), encoded by ADGRG1, is an adhesion GPCR found on diverse cell types, including neural progenitor cells, melanoma cells, and lymphocytes, such as effector memory T cells, γδ T cells, and NK cells. Using RNA-sequencing and high-resolution flow cytometry, we found that GPR56 mRNA and protein expression increased with NK cell differentiation, reaching its peak in adaptive NK cells. Small interfering RNA silencing of GPR56 led to increased spontaneous and chemokine-induced migration, suggesting that GPR56 functions as an upstream checkpoint for migration of highly differentiated NK cells. Increased NK cell migration could also be induced by agonistic stimulation of GPR56 leading to rapid internalization and deactivation of the receptor. Mechanistically, GPR56 ligation and downregulation were associated with transcriptional coactivator with PDZ-binding motif translocation to the nucleus and increased actin polymerization. Together, these data provide insights into the role of GPR56 in the migratory behavior of human NK cell subsets and may open possibilities to improve NK cell infiltration into cancer tissues by releasing a migratory checkpoint.

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来源期刊
Journal of immunology
Journal of immunology 医学-免疫学
CiteScore
8.20
自引率
2.30%
发文量
495
审稿时长
1 months
期刊介绍: The JI publishes novel, peer-reviewed findings in all areas of experimental immunology, including innate and adaptive immunity, inflammation, host defense, clinical immunology, autoimmunity and more. Special sections include Cutting Edge articles, Brief Reviews and Pillars of Immunology. The JI is published by The American Association of Immunologists (AAI)
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