RNA 受体 RIG-I 结合合成寡聚脱氧核苷酸可促进肺炎存活。

IF 6.3 1区 医学 Q1 MEDICINE, RESEARCH & EXPERIMENTAL JCI insight Pub Date : 2024-11-08 DOI:10.1172/jci.insight.180584
Yongxing Wang, Vikram V Kulkarni, Jezreel PantaleónGarcía, Michael K Longmire, Mathilde Lethier, Stephen Cusack, Scott E Evans
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引用次数: 0

摘要

肺炎是全球公共卫生的一大威胁,需要新的预防和治疗策略。肺上皮细胞是一个关键的环境界面,它既是病原体入侵的物理屏障,又能主动感知和应对病原体。我们曾报道,用一种由二乙酰化脂肽和合成 CpG 寡脱氧核苷酸(ODN)组成的组合疗法刺激肺上皮细胞,可诱导协同性肺炎保护,抵御多种病原体的侵袭。我们在此报告,缺乏 Toll 样受体 9 (TLR9) 的小鼠(以前曾描述过 ODN 的受体)仍显示出部分 ODN 诱导的保护作用。这促使我们寻找另一种 ODN 受体,我们通过质谱发现 RNA 传感器 RIG-I 也能结合 DNA 样的 ODN。RIG-I 与 ODN 的结合导致了 MAVS 依赖性肺炎保护信号事件。虽然 RIG-I 对本地防御病毒感染至关重要,但我们报告说,用 ODN 激活治疗性 RIG-I 能在病毒和细菌挑战后促进病原体杀灭和宿主存活。这些数据表明,ODN 诱导的最大肺炎保护需要同时激活 TLR9/MyD88 和 RIG-I/MAVS 信号通路。这些发现不仅确定了我们认为是 DNA 类分子的新型模式识别受体,而且揭示了一种潜在的治疗策略,可在易感性高峰期保护易感者免受致命性肺炎的侵害。
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The RNA receptor RIG-I binding synthetic oligodeoxynucleotide promotes pneumonia survival.

Pneumonia is a worldwide threat to public health, demanding novel preventative and therapeutic strategies. The lung epithelium is a critical environmental interface that functions as a physical barrier to pathogen invasion while also actively sensing and responding to pathogens. We have reported that stimulating lung epithelial cells with a combination therapeutic consisting of a diacylated lipopeptide and a synthetic CpG oligodeoxynucleotide (ODN) induces synergistic pneumonia protection against a wide range of pathogens. We report here that mice deficient in TLR9, the previously described receptor for ODN, still displayed partial ODN-induced protection. This prompted us to seek an alternate ODN receptor, and we discovered by mass spectroscopy that the RNA sensor RIG-I could also bind DNA-like ODN. ODN binding by RIG-I resulted in MAVS-dependent pneumonia-protective signaling events. While RIG-I is essential to native defenses against viral infections, we report that therapeutic RIG-I activation with ODN promoted pathogen killing and host survival following both viral and bacterial challenges. These data indicate that maximal ODN-induced pneumonia protection requires activation of both the TLR9/MyD88 and RIG-I/MAVS signaling pathways. These findings not only identify what we believe to be a novel pattern recognition receptor for DNA-like molecules, but reveal a potential therapeutic strategy to protect susceptible individuals against lethal pneumonias during periods of peak vulnerability.

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来源期刊
JCI insight
JCI insight Medicine-General Medicine
CiteScore
13.70
自引率
1.20%
发文量
543
审稿时长
6 weeks
期刊介绍: JCI Insight is a Gold Open Access journal with a 2022 Impact Factor of 8.0. It publishes high-quality studies in various biomedical specialties, such as autoimmunity, gastroenterology, immunology, metabolism, nephrology, neuroscience, oncology, pulmonology, and vascular biology. The journal focuses on clinically relevant basic and translational research that contributes to the understanding of disease biology and treatment. JCI Insight is self-published by the American Society for Clinical Investigation (ASCI), a nonprofit honor organization of physician-scientists founded in 1908, and it helps fulfill the ASCI's mission to advance medical science through the publication of clinically relevant research reports.
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