David Allan, James Tooby, Lindsay Starling, Ross Tucker, Éanna C Falvey, Danielle M Salmon, James Brown, Sam Hudson, Keith A Stokes, Ben Jones, Simon P T Kemp, Patrick O'Halloran, Matt Cross, Gregory Tierney
{"title":"精英级别男子和女子橄榄球联盟比赛中与头部加速事件相关的球员和比赛特征。","authors":"David Allan, James Tooby, Lindsay Starling, Ross Tucker, Éanna C Falvey, Danielle M Salmon, James Brown, Sam Hudson, Keith A Stokes, Ben Jones, Simon P T Kemp, Patrick O'Halloran, Matt Cross, Gregory Tierney","doi":"10.1136/bmjsem-2024-001954","DOIUrl":null,"url":null,"abstract":"<p><strong>Objective: </strong>To examine the likelihood of head acceleration events (HAEs) as a function of previously identified risk factors: match time, player status (starter or substitute) and pitch location in elite-level men's and women's rugby union matches.</p><p><strong>Methods: </strong>Instrumented mouthguard data were collected from 179 and 107 players in the men's and women's games and synchronised to video-coded match footage. Head peak resultant linear acceleration (PLA) and peak resultant angular acceleration were extracted from each HAE. Field location was determined for HAEs linked to a tackle, carry or ruck. HAE incidence was calculated per player hour across PLA recording thresholds with 95% CIs estimated. Propensity was calculated as the percentage of contact events that caused HAEs across PLA recording thresholds, with a 95% CI estimated. Significance was assessed by non-overlapping 95% CIs.</p><p><strong>Results: </strong>29 099 and 6277 HAEs were collected from 1214 and 577 player-matches in the men's and women's games. No significant differences in match quarter HAE incidence or propensity were found. Substitutes had higher HAE incidence than starters at lower PLA recording thresholds for men but similar HAE propensity. HAEs were more likely to occur in field locations with high contact event occurrence.</p><p><strong>Conclusion: </strong>Strategies to reduce HAE incidence need not consider match time or status as a substitute or starter as HAE rates are similar throughout matches, without differences in propensity between starters and substitutes. HAE incidence is proportional to contact frequency, and strategies that reduce either frequency or propensity for contact to cause head contact may be explored.</p>","PeriodicalId":47417,"journal":{"name":"BMJ Open Sport & Exercise Medicine","volume":"10 4","pages":"e001954"},"PeriodicalIF":3.9000,"publicationDate":"2024-10-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11459297/pdf/","citationCount":"0","resultStr":"{\"title\":\"Player and match characteristics associated with head acceleration events in elite-level men's and women's rugby union matches.\",\"authors\":\"David Allan, James Tooby, Lindsay Starling, Ross Tucker, Éanna C Falvey, Danielle M Salmon, James Brown, Sam Hudson, Keith A Stokes, Ben Jones, Simon P T Kemp, Patrick O'Halloran, Matt Cross, Gregory Tierney\",\"doi\":\"10.1136/bmjsem-2024-001954\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><strong>Objective: </strong>To examine the likelihood of head acceleration events (HAEs) as a function of previously identified risk factors: match time, player status (starter or substitute) and pitch location in elite-level men's and women's rugby union matches.</p><p><strong>Methods: </strong>Instrumented mouthguard data were collected from 179 and 107 players in the men's and women's games and synchronised to video-coded match footage. Head peak resultant linear acceleration (PLA) and peak resultant angular acceleration were extracted from each HAE. Field location was determined for HAEs linked to a tackle, carry or ruck. HAE incidence was calculated per player hour across PLA recording thresholds with 95% CIs estimated. Propensity was calculated as the percentage of contact events that caused HAEs across PLA recording thresholds, with a 95% CI estimated. Significance was assessed by non-overlapping 95% CIs.</p><p><strong>Results: </strong>29 099 and 6277 HAEs were collected from 1214 and 577 player-matches in the men's and women's games. No significant differences in match quarter HAE incidence or propensity were found. Substitutes had higher HAE incidence than starters at lower PLA recording thresholds for men but similar HAE propensity. 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引用次数: 0
摘要
目的研究在精英级别的男子和女子橄榄球联盟比赛中,头部加速事件(HAE)发生的可能性与之前确定的风险因素(比赛时间、球员状态(首发或替补)和场地位置)之间的函数关系:方法:在男子和女子比赛中,分别从 179 名和 107 名球员身上收集了护齿器数据,并与视频编码的比赛录像同步。从每个 HAE 中提取头部峰值结果线性加速度 (PLA) 和峰值结果角加速度。对于与擒抱、搬运或拦截相关的 HAE,则确定其现场位置。根据PLA记录阈值计算每名球员每小时的HAE发生率,并估算95% CIs。倾向性计算方法是,根据 PLA 记录阈值,计算引起 HAE 的接触事件的百分比,并估算 95% CI。结果:分别从 1214 场和 577 场男子和女子比赛中收集到 29 099 次和 6277 次 HAE。在比赛季度 HAE 发生率或倾向方面没有发现明显差异。在较低的 PLA 记录阈值下,替补球员的 HAE 发生率高于首发球员,但 HAE 发生倾向相似。HAE更有可能发生在接触事件高发的赛场:减少 HAE 发生率的策略无需考虑比赛时间或替补或首发身份,因为 HAE 发生率在整场比赛中相似,首发和替补之间的倾向性也没有差异。HAE 的发生率与接触频率成正比,因此可以探索降低接触频率或接触导致头部接触的倾向的策略。
Player and match characteristics associated with head acceleration events in elite-level men's and women's rugby union matches.
Objective: To examine the likelihood of head acceleration events (HAEs) as a function of previously identified risk factors: match time, player status (starter or substitute) and pitch location in elite-level men's and women's rugby union matches.
Methods: Instrumented mouthguard data were collected from 179 and 107 players in the men's and women's games and synchronised to video-coded match footage. Head peak resultant linear acceleration (PLA) and peak resultant angular acceleration were extracted from each HAE. Field location was determined for HAEs linked to a tackle, carry or ruck. HAE incidence was calculated per player hour across PLA recording thresholds with 95% CIs estimated. Propensity was calculated as the percentage of contact events that caused HAEs across PLA recording thresholds, with a 95% CI estimated. Significance was assessed by non-overlapping 95% CIs.
Results: 29 099 and 6277 HAEs were collected from 1214 and 577 player-matches in the men's and women's games. No significant differences in match quarter HAE incidence or propensity were found. Substitutes had higher HAE incidence than starters at lower PLA recording thresholds for men but similar HAE propensity. HAEs were more likely to occur in field locations with high contact event occurrence.
Conclusion: Strategies to reduce HAE incidence need not consider match time or status as a substitute or starter as HAE rates are similar throughout matches, without differences in propensity between starters and substitutes. HAE incidence is proportional to contact frequency, and strategies that reduce either frequency or propensity for contact to cause head contact may be explored.