胆汁酸是α-突触核蛋白聚合的调节剂:对帕金森病治疗的意义。

IF 4.1 3区 医学 Q2 BIOCHEMISTRY & MOLECULAR BIOLOGY ACS Chemical Neuroscience Pub Date : 2024-11-06 Epub Date: 2024-10-15 DOI:10.1021/acschemneuro.4c00459
Harpreet Kaur, Devansh Swadia, Sharmistha Sinha
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引用次数: 0

摘要

帕金森病(PD)是一种神经退行性疾病,其特征是α-突触核蛋白聚集成有毒的淀粉样纤维。最新研究表明,帕金森病患者体内胆汁酸的改变可能会影响其聚集。本研究调查了石胆酸(LCA)和脱氧胆酸(DCA)对α-突触核蛋白聚集和毒性的影响。LCA 能明显加速聚集,将滞后期缩短 75%,而 DCA 的影响较小,将滞后期缩短 30%。结合研究表明,LCA 与α-突触核蛋白的 NAC 区域相互作用,而 DCA 则与α-突触核蛋白的 N 端区域相互作用。聚合试验和电泳显示,LCA 比 DCA 更有效地促进有毒的抗 SDS 寡聚体的形成。细胞毒性检测证实,LCA处理的样本中细胞存活率较低。此外,LCA 和 DCA 联合处理会导致聚集和毒性增强,显示出协同效应。这些发现突显了胆汁酸在α-突触核蛋白聚集和帕金森病发病机制中的作用,表明针对胆汁酸代谢的研究可能是治疗帕金森病的一种策略。
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Bile Acids as Modulators of α-Synuclein Aggregation: Implications for Parkinson's Therapy.

Parkinson's disease (PD) is a neurodegenerative disorder characterized by the aggregation of α-synuclein into toxic amyloid fibrils. Recent research suggests that bile acids altered in PD may influence their aggregation. This study investigates the effects of lithocholic acid (LCA) and deoxycholic acid (DCA) on α-synuclein aggregation and toxicity. LCA significantly accelerates aggregation, reducing the lag phase by 75%, while DCA has a milder impact, decreasing the lag phase by 30%. Binding studies show that LCA interacts with the NAC region and DCA with the N-terminal region of α-synuclein. Aggregation assays and electrophoresis reveal that LCA promotes the formation of toxic, SDS-resistant oligomers more effectively than DCA. Cytotoxicity assays confirm a lower cell viability in LCA-treated samples. Additionally, combined LCA and DCA treatment results in enhanced aggregation and toxicity, indicating a synergistic effect. These findings highlight the role of bile acids in α-synuclein aggregation and PD pathogenesis, suggesting that targeting bile acid metabolism could be a therapeutic strategy for PD.

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来源期刊
ACS Chemical Neuroscience
ACS Chemical Neuroscience BIOCHEMISTRY & MOLECULAR BIOLOGY-CHEMISTRY, MEDICINAL
CiteScore
9.20
自引率
4.00%
发文量
323
审稿时长
1 months
期刊介绍: ACS Chemical Neuroscience publishes high-quality research articles and reviews that showcase chemical, quantitative biological, biophysical and bioengineering approaches to the understanding of the nervous system and to the development of new treatments for neurological disorders. Research in the journal focuses on aspects of chemical neurobiology and bio-neurochemistry such as the following: Neurotransmitters and receptors Neuropharmaceuticals and therapeutics Neural development—Plasticity, and degeneration Chemical, physical, and computational methods in neuroscience Neuronal diseases—basis, detection, and treatment Mechanism of aging, learning, memory and behavior Pain and sensory processing Neurotoxins Neuroscience-inspired bioengineering Development of methods in chemical neurobiology Neuroimaging agents and technologies Animal models for central nervous system diseases Behavioral research
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