Wnt/β-catenin信号通路中的eIF3a翻译控制轴与结肠肿瘤发生。

IF 9.1 1区 医学 Q1 ONCOLOGY Cancer letters Pub Date : 2024-10-15 DOI:10.1016/j.canlet.2024.217303
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引用次数: 0

摘要

蛋白质合成中的转化起始是基因表达的重要调控步骤,其失调可能导致癌症等疾病。eIF4E/4E-BP 的翻译控制已被深入研究,并在各种生物过程中对 mTOR 信号转导做出了贡献。在这里,我们报告了结肠肿瘤发生过程中 Wnt/β-catenin 信号通路中一个新的转译控制轴,即肿瘤发生和预后的阴阳因子 eIF3a。我们发现,eIF3a在人类结肠癌组织、癌前腺瘤息肉中表达上调,并与人类样本中的β-catenin水平和APC突变相关,eIF3a的过表达可转化肠上皮细胞。我们还发现,eIF3a的表达受Wnt/β-catenin信号通路调控,其启动子中有一个活跃的TCF/LEF结合位点,敲除eIF3a可抑制APC突变诱导的APCmin/+小鼠自发性结肠肿瘤发生。综上所述,我们得出结论:eIF3a在结肠癌中的上调是由于APC突变引起的,它通过在Wnt/β-catenin信号通路中增加一个翻译控制轴参与结肠癌的发生,可以作为结肠癌干预的潜在靶点。
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The eIF3a translational control axis in the Wnt/β-catenin signaling pathway and colon tumorigenesis
Translational initiation in protein synthesis is an important regulatory step in gene expression and its dysregulation may result in diseases such as cancer. Translational control by eIF4E/4E-BP has been well studied and contributes to mTOR signaling in various biological processes. Here, we report a novel translational control axis in the Wnt/β-catenin signaling pathway in colon tumorigenesis by eIF3a, a Yin-Yang factor in tumorigenesis and prognosis. We show that eIF3a expression is upregulated in human colon cancer tissues, pre-cancerous adenoma polyps, and associates with β-catenin level and APC mutation in human samples, and that eIF3a overexpression transforms intestinal epithelial cells. We also show that eIF3a expression is regulated by the Wnt/β-catenin signaling pathway with an active TCF/LEF binding site in its promoter and that eIF3a knockdown inhibits APC mutation-induced spontaneous colon tumorigenesis in APCmin/+ mice. Together, we conclude that eIF3a upregulation in colon cancer is due to APC mutation and it participates in colon tumorigenesis by adding a translational control axis in the Wnt/β-catenin signaling pathway and that it can serve as a potential target for colon cancer intervention.
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来源期刊
Cancer letters
Cancer letters 医学-肿瘤学
CiteScore
17.70
自引率
2.10%
发文量
427
审稿时长
15 days
期刊介绍: Cancer Letters is a reputable international journal that serves as a platform for significant and original contributions in cancer research. The journal welcomes both full-length articles and Mini Reviews in the wide-ranging field of basic and translational oncology. Furthermore, it frequently presents Special Issues that shed light on current and topical areas in cancer research. Cancer Letters is highly interested in various fundamental aspects that can cater to a diverse readership. These areas include the molecular genetics and cell biology of cancer, radiation biology, molecular pathology, hormones and cancer, viral oncology, metastasis, and chemoprevention. The journal actively focuses on experimental therapeutics, particularly the advancement of targeted therapies for personalized cancer medicine, such as metronomic chemotherapy. By publishing groundbreaking research and promoting advancements in cancer treatments, Cancer Letters aims to actively contribute to the fight against cancer and the improvement of patient outcomes.
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