Oritoke M Okeowo, Victor E Anadu, Olayemi K Ijomone, Michael Aschner, Omamuyovwi M Ijomone
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Thereafter, an immunohistochemical procedure was utilised to evaluate neurochemical markers of glial function, myelination, oxidative stress, and apoptosis in the hippocampus, prefrontal cortex (PFC), and striatum. Results showed that stress and metal exposure increased oxidative stress markers and reduced antioxidant levels. Further, combined stress and metal exposure reduced various forms of learning and memory ability in rats. In addition, there were alterations in Iba1 activity and Nrf2 levels, reduced Olig2 and myelin basic protein (MBP) levels, and increased caspase-3 expression. These neurotoxic outcomes were mostly exacerbated by co-exposure to stress and metals. 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引用次数: 0
摘要
重金属(锰和镍)的积累和长期暴露于压力下与不良健康后果有关。多项研究表明,压力和金属暴露对大脑功能有影响。然而,还没有研究探讨同时暴露于压力、锰和镍对大脑的影响。本研究通过评估大鼠模型中的氧化和神经胶质反应、细胞凋亡活动以及认知过程,填补了这一空白。成年 Wistar 大鼠分别暴露于载体(对照组)、束缚应激、25 毫克/千克的锰(Mn)或镍(Ni),或联合束缚应激加 Mn 或 Ni。治疗后,对大鼠进行了几种行为范式来评估认知功能。对酶活性和 ATP 酶水平进行了评估。之后,利用免疫组化程序评估了海马、前额叶皮质和纹状体中神经胶质功能、髓鞘化、氧化应激和细胞凋亡的神经化学标记物。结果表明,压力和金属暴露增加了氧化应激标记物,降低了抗氧化剂水平。此外,压力和金属的联合暴露降低了大鼠各种形式的学习和记忆能力。此外,Iba1活性和Nrf2水平也发生了改变,Olig2和髓鞘碱性蛋白(MBP)水平降低,Caspase-3表达增加。这些神经毒性结果在同时暴露于应激和金属时大多会加剧。总之,我们的研究结果表明,应激和金属暴露会损害认知能力,诱导氧化应激和细胞凋亡,并导致脱髓鞘效应,而应激和金属的联合暴露会使脱髓鞘效应更加严重。
Combined Restraint Stress and Metal Exposure Paradigms in Rats: Unravelling Behavioural and Neurochemical Perturbations.
Accumulation of heavy metals (Mn and Ni) and prolonged exposure to stress are associated with adverse health outcomes. Various studies have shown the impacts of stress and metal exposures on brain function. However, no study has examined the effects of co-exposure to stress, Mn, and Ni on the brain. This study addresses this gap by evaluating oxidative and glial responses, apoptotic activity, as well as cognitive processes in a rat model. Adult Wistar rats were exposed to vehicle (control), restraint stress, 25 mg/kg of manganese (Mn) or nickel (Ni), or combined restraint stress plus Mn or Ni. Following treatment, rats were subjected to several behavioural paradigms to assess cognitive function. Enzyme activity, as well as ATPase levels, were evaluated. Thereafter, an immunohistochemical procedure was utilised to evaluate neurochemical markers of glial function, myelination, oxidative stress, and apoptosis in the hippocampus, prefrontal cortex (PFC), and striatum. Results showed that stress and metal exposure increased oxidative stress markers and reduced antioxidant levels. Further, combined stress and metal exposure reduced various forms of learning and memory ability in rats. In addition, there were alterations in Iba1 activity and Nrf2 levels, reduced Olig2 and myelin basic protein (MBP) levels, and increased caspase-3 expression. These neurotoxic outcomes were mostly exacerbated by co-exposure to stress and metals. Overall, our findings establish that stress and metal exposures impaired cognitive performance, induced oxidative stress and apoptosis, and led to demyelination effects which were worsened by combined stress and metal exposure.
期刊介绍:
Molecular Neurobiology is an exciting journal for neuroscientists needing to stay in close touch with progress at the forefront of molecular brain research today. It is an especially important periodical for graduate students and "postdocs," specifically designed to synthesize and critically assess research trends for all neuroscientists hoping to stay active at the cutting edge of this dramatically developing area. This journal has proven to be crucial in departmental libraries, serving as essential reading for every committed neuroscientist who is striving to keep abreast of all rapid developments in a forefront field. Most recent significant advances in experimental and clinical neuroscience have been occurring at the molecular level. Until now, there has been no journal devoted to looking closely at this fragmented literature in a critical, coherent fashion. Each submission is thoroughly analyzed by scientists and clinicians internationally renowned for their special competence in the areas treated.