Ptk6-/- 雄性小鼠小肠中琥珀酸诱导的 2 型免疫激活和分泌细胞生成受损。

IF 8.1 1区 生物学 Q1 CELL BIOLOGY Cell Death & Disease Pub Date : 2024-10-26 DOI:10.1038/s41419-024-07149-9
Katarina Vlajic, Wenjun Bie, Milica B Gilic, Angela L Tyner
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引用次数: 0

摘要

蛋白酪氨酸激酶 6(PTK6)是一种细胞内酪氨酸激酶,与酪氨酸激酶 SRC 家族关系密切。它在上皮内衬中表达,调节肠上皮的再生和修复。对公开数据集的分析表明,当2型免疫激活时,Ptk6在簇细胞中上调。我们发现,干扰 Ptk6 会影响参与肠道免疫反应的基因表达。在雄性而非雌性小鼠体内施用琥珀酸(模拟感染并激活簇细胞)会发现先天性免疫反应的激活依赖于 PTK6。与所有野生型小鼠和Ptk6-/-雌性小鼠相反,Ptk6-/-雄性小鼠在琥珀酸处理后不会激活先天性免疫或上调簇细胞和上睑分泌细胞系的分化。从机理上讲,我们发现 PTK6 在雄性小鼠而非雌性小鼠的肠道器官组织中调控簇细胞效应功能和 2 型先天性免疫激活所需的基因 Il25 和 Irag2。在克罗恩病患者中,PTK6 在肠道非炎症区域的簇细胞中上调。这些数据强调了 PTK6 在肠道先天性免疫性别差异中的作用,并为 IL-25 的调控提供了新的见解。
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Impaired activation of succinate-induced type 2 immunity and secretory cell production in the small intestines of Ptk6-/- male mice.

Protein tyrosine kinase 6 (PTK6) is an intracellular tyrosine kinase that is distantly related to the SRC family of tyrosine kinases. It is expressed in epithelial linings and regulates regeneration and repair of the intestinal epithelium. Analysis of publicly available datasets showed Ptk6 is upregulated in tuft cells upon activation of type 2 immunity. We found that disruption of Ptk6 influences gene expression involved in intestinal immune responses. Administration of succinate, which mimics infection and activates tuft cells, revealed PTK6-dependent activation of innate immune responses in male but not female mice. In contrast to all wild type and Ptk6-/- female mice, Ptk6-/- male mice do not activate innate immunity or upregulate differentiation of the tuft and goblet secretory cell lineages following succinate treatment. Mechanistically, we found that PTK6 regulates Il25 and Irag2, genes that are required for tuft cell effector functions and activation of type 2 innate immunity, in organoids derived from intestines of male but not female mice. In patients with Crohn's disease, PTK6 is upregulated in tuft cells in noninflamed regions of intestine. These data highlight roles for PTK6 in contributing to sex differences in intestinal innate immunity and provide new insights into the regulation of IL-25.

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来源期刊
Cell Death & Disease
Cell Death & Disease CELL BIOLOGY-
CiteScore
15.10
自引率
2.20%
发文量
935
审稿时长
2 months
期刊介绍: Brought to readers by the editorial team of Cell Death & Differentiation, Cell Death & Disease is an online peer-reviewed journal specializing in translational cell death research. It covers a wide range of topics in experimental and internal medicine, including cancer, immunity, neuroscience, and now cancer metabolism. Cell Death & Disease seeks to encompass the breadth of translational implications of cell death, and topics of particular concentration will include, but are not limited to, the following: Experimental medicine Cancer Immunity Internal medicine Neuroscience Cancer metabolism
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