抗酶的蛋白质降解取决于 N 端脱胶子。

IF 4.5 3区 生物学 Q1 BIOCHEMISTRY & MOLECULAR BIOLOGY Protein Science Pub Date : 2024-11-01 DOI:10.1002/pro.5199
Ju-Yi Hsieh, Pui-Ying Leong, Yi-Fang Yang, Yi-Liang Liu, Guang-Yaw Liu, Hui-Chih Hung
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引用次数: 0

摘要

抗酶(AZ)是一种调节蛋白,在调节鸟氨酸脱羧酶(ODC)的活性方面起着至关重要的作用,而鸟氨酸脱羧酶是多胺生物合成复杂途径中的初始酶和限速酶。AZ 可促进 ODC 的快速降解,从而调节细胞多胺的水平。这项研究揭示了一种新的不依赖泛素的 AZ 降解机制,强调了 N 端脱胶子的重要作用。与传统的泛素依赖性降解相反,我们的研究结果表明,AZ 的降解受其 N 端区域的显著影响。通过体外降解实验、环己亚胺追逐实验、差示扫描量热法和多胺细胞浓度测量等一系列实验,我们证明了 N 端截短可显著增强 AZ 的稳定性,并通过加速 ODC 降解来促进多胺水平的降低。去除 AZ 的 N 端部分可降低降解率,增强蛋白质的热稳定性,从而更有效地抑制多胺的合成。对 AZ 异构体 AZ1、AZ2 和 AZ3 的分析证实了这些发现,它们根据特定的 N 端片段显示出不同的降解模式。这证实了一种独立于赖氨酸泛素化的降解机制,即由作为降解子的内在紊乱 N 端区域驱动的降解。这些结果强调了 N 端结构域在 AZ 活性和维持多胺平衡方面的重要调节功能。
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Protein degradation of antizyme depends on the N-terminal degrons.

Antizyme (AZ) is a regulatory protein that plays a crucial role in modulating the activity of ornithine decarboxylase (ODC), which is the initial and rate-limiting enzyme in the complex pathway of polyamine biosynthesis. AZ facilitates the swift degradation of ODC, thereby modulating the levels of cellular polyamines. This study unveils a new ubiquitin-independent mechanism for AZ degradation, emphasizing the essential role of N-terminal degrons. Contrary to traditional ubiquitin-dependent degradation, our findings reveal that AZ degradation is significantly influenced by its N-terminal region. By conducting a series of experiments, including in vitro degradation assays, cycloheximide chase experiments, differential scanning calorimetry, and measurement of cellular concentrations of polyamines, we demonstrate that N-terminal truncation significantly enhances AZ's stability and facilitates the reduction of polyamine levels by accelerating ODC degradation. The removal of the N-terminal portion of AZ results in a reduced degradation rate and enhanced thermal stability of the protein, leading to a more efficient inhibition of polyamine synthesis. These findings are corroborated by the analysis of AZ isoforms, AZ1, AZ2, and AZ3, which display differential degradation patterns based on the specific N-terminal segments. This substantiates a degradation mechanism driven by an intrinsically disordered N-terminal region acting as a degron, independent of lysine ubiquitination. These results underscore the significant regulatory function of the N-terminal domain in the activity of AZ and the maintenance of polyamine homeostasis.

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来源期刊
Protein Science
Protein Science 生物-生化与分子生物学
CiteScore
12.40
自引率
1.20%
发文量
246
审稿时长
1 months
期刊介绍: Protein Science, the flagship journal of The Protein Society, is a publication that focuses on advancing fundamental knowledge in the field of protein molecules. The journal welcomes original reports and review articles that contribute to our understanding of protein function, structure, folding, design, and evolution. Additionally, Protein Science encourages papers that explore the applications of protein science in various areas such as therapeutics, protein-based biomaterials, bionanotechnology, synthetic biology, and bioelectronics. The journal accepts manuscript submissions in any suitable format for review, with the requirement of converting the manuscript to journal-style format only upon acceptance for publication. Protein Science is indexed and abstracted in numerous databases, including the Agricultural & Environmental Science Database (ProQuest), Biological Science Database (ProQuest), CAS: Chemical Abstracts Service (ACS), Embase (Elsevier), Health & Medical Collection (ProQuest), Health Research Premium Collection (ProQuest), Materials Science & Engineering Database (ProQuest), MEDLINE/PubMed (NLM), Natural Science Collection (ProQuest), and SciTech Premium Collection (ProQuest).
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