ZBTB18 可调节细胞因子的表达,并影响胶质母细胞瘤中小胶质细胞/巨噬细胞的招募和承诺。

IF 5.2 1区 生物学 Q1 BIOLOGY Communications Biology Pub Date : 2024-11-08 DOI:10.1038/s42003-024-07144-y
Roberto Ferrarese, Kevin Joseph, Geoffroy Andrieux, Ira Verena Haase, Francesca Zanon, Eva Kling, Annalisa Izzo, Eyleen Corrales, Marius Schwabenland, Marco Prinz, Vidhya Madapusi Ravi, Melanie Boerries, Dieter Henrik Heiland, Maria Stella Carro
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引用次数: 0

摘要

胶质瘤相关巨噬细胞/小胶质细胞(GAMs)在胶质母细胞瘤(GBM)的发展过程中扮演着重要角色,因为它们会被大量招募到肿瘤部位,并极化为肿瘤促进表型。GAMs 能分泌多种细胞因子,促进肿瘤细胞的生长和侵袭,并阻止其他免疫细胞对肿瘤做出免疫反应。在这里,我们证明了锌指和含 BTB 结构域 18(ZBTB18)--一种在胶质母细胞瘤中具有肿瘤抑制功能的转录抑制因子--会影响关键细胞因子的产生,而关键细胞因子对 GAMs 起着趋化吸引作用。同样,在细胞培养和体内实验中,我们观察到当胶质母细胞瘤细胞表达 ZBTB18 时,胶质母细胞瘤的迁移减少。此外,RNA 测序分析表明,胶质母细胞瘤细胞中 ZBTB18 的存在改变了条件小胶质细胞的承诺,表明其丧失了免疫抑制表型,获得了促炎特征。因此,增加胶质母细胞瘤细胞中 ZBTB18 表达的治疗方法可能是胶质母细胞瘤免疫治疗的有效辅助手段。
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ZBTB18 regulates cytokine expression and affects microglia/macrophage recruitment and commitment in glioblastoma.

Glioma associated macrophages/microglia (GAMs) play an important role in glioblastoma (GBM) progression, due to their massive recruitment to the tumor site and polarization to a tumor promoting phenotype. GAMs secrete a variety of cytokines, which facilitate tumor cell growth and invasion, and prevent other immune cells from mounting an immune response against the tumor. Here, we demonstrate that zinc finger and BTB containing domain 18 (ZBTB18), a transcriptional repressor with tumor suppressive function in glioblastoma, impairs the production of key cytokines, which function as chemoattractant for GAMs. Consistently, we observe a reduced migration of GAMs when ZBTB18 is expressed by glioblastoma cells, both in cell culture and in vivo experiments. Moreover, RNA sequencing analysis shows that the presence of ZBTB18 in glioblastoma cells alters the commitment of conditioned microglia, suggesting the loss of the immune-suppressive phenotype and the acquisition of pro-inflammatory features. Thus, therapeutic approaches to increase ZBTB18 expression in GBM cells could represent an effective adjuvant to immune therapy in GBM.

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来源期刊
Communications Biology
Communications Biology Medicine-Medicine (miscellaneous)
CiteScore
8.60
自引率
1.70%
发文量
1233
审稿时长
13 weeks
期刊介绍: Communications Biology is an open access journal from Nature Research publishing high-quality research, reviews and commentary in all areas of the biological sciences. Research papers published by the journal represent significant advances bringing new biological insight to a specialized area of research.
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