NOTCH3 通过直接调节 SPP1 和激活 PI3K/AKT 通路促进膀胱癌的恶性进展。

IF 8.1 1区 生物学 Q1 CELL BIOLOGY Cell Death & Disease Pub Date : 2024-11-18 DOI:10.1038/s41419-024-07241-0
Changxue Liu, Huaixi Ge, Chengquan Shen, Ding Hu, Xinzhao Zhao, Ruize Qin, Yonghua Wang
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引用次数: 0

摘要

Notch受体3(NOTCH3)在膀胱癌(BLCA)恶性进展中的生物学作用和确切的分子机制仍不清楚。本研究发现,NOTCH3在膀胱癌患者中明显上调,并与不良预后相关。功能实验表明,NOTCH3 基因敲除可抑制 BLCA 细胞的增殖、迁移和侵袭,并显著抑制肿瘤在体内的生长和转移。染色质免疫沉淀和双荧光素酶报告实验证实,NOTCH3可通过与SPP1启动子中的CSL元件结合,促进NOTCH3潜在下游靶基因分泌磷蛋白1(SPP1)的转录。此外,我们还发现,靶向 NOTCH3 可通过抑制 SPP1-PI3K/AKT 轴来抑制 BLCA 的生长和转移。我们的研究强调了NOTCH3-SPP1-PI3K/AKT轴在BLCA恶性进展中的关键作用,提示NOTCH3可能是BLCA的潜在治疗靶点。
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NOTCH3 promotes malignant progression of bladder cancer by directly regulating SPP1 and activating PI3K/AKT pathway.

The biological role and precise molecular mechanisms of Notch receptor 3 (NOTCH3) in the malignant progression of bladder cancer (BLCA) remain unclear. In this study, we found that NOTCH3 was significantly upregulated and associated with poor prognosis in BLCA patients. Functional experiments demonstrated that NOTCH3 knockdown inhibited BLCA cell proliferation, migration, invasion and significantly suppressed tumor growth and metastasis in vivo as well. Mechanically, chromatin immunoprecipitation and dual-luciferase reporter assays confirmed that NOTCH3 could promote the transcription of secreted phosphoprotein 1 (SPP1), a potential downstream target gene of NOTCH3, by binding to the CSL elements in the SPP1 promoter. Moreover, we also found that targeting NOTCH3 inhibited BLCA growth and metastasis by suppressing the SPP1-PI3K/AKT axis. Our study highlights the critical role of NOTCH3-SPP1-PI3K/AKT axis in the malignant progression of BLCA, suggesting that NOTCH3 may be a potential therapeutic target for BLCA.

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来源期刊
Cell Death & Disease
Cell Death & Disease CELL BIOLOGY-
CiteScore
15.10
自引率
2.20%
发文量
935
审稿时长
2 months
期刊介绍: Brought to readers by the editorial team of Cell Death & Differentiation, Cell Death & Disease is an online peer-reviewed journal specializing in translational cell death research. It covers a wide range of topics in experimental and internal medicine, including cancer, immunity, neuroscience, and now cancer metabolism. Cell Death & Disease seeks to encompass the breadth of translational implications of cell death, and topics of particular concentration will include, but are not limited to, the following: Experimental medicine Cancer Immunity Internal medicine Neuroscience Cancer metabolism
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