更正:[肺动脉内皮细胞在 EET 诱导下的增殖、存活和血管生成需要 JNK/c-Jun 的激活]。

IF 5 2区 医学 Q1 BIOCHEMISTRY & MOLECULAR BIOLOGY Journal of Lipid Research Pub Date : 2024-11-18 DOI:10.1016/j.jlr.2024.100691
Jun Ma, Lei Zhang, Weina Han, Tingting Shen, Cui Ma, Yun Liu, Xiaowei Nie, Mengmeng Liu, Yajuan Ran, Daling Zhu
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Corrigendum to: [Activation of JNK/c-Jun is required for the proliferation, survival, and angiogenesis induced by EET in pulmonary artery endothelial cells].
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来源期刊
Journal of Lipid Research
Journal of Lipid Research 生物-生化与分子生物学
CiteScore
11.10
自引率
4.60%
发文量
146
审稿时长
41 days
期刊介绍: The Journal of Lipid Research (JLR) publishes original articles and reviews in the broadly defined area of biological lipids. We encourage the submission of manuscripts relating to lipids, including those addressing problems in biochemistry, molecular biology, structural biology, cell biology, genetics, molecular medicine, clinical medicine and metabolism. Major criteria for acceptance of articles are new insights into mechanisms of lipid function and metabolism and/or genes regulating lipid metabolism along with sound primary experimental data. Interpretation of the data is the authors’ responsibility, and speculation should be labeled as such. Manuscripts that provide new ways of purifying, identifying and quantifying lipids are invited for the Methods section of the Journal. JLR encourages contributions from investigators in all countries, but articles must be submitted in clear and concise English.
期刊最新文献
Corrigendum to: [Activation of JNK/c-Jun is required for the proliferation, survival, and angiogenesis induced by EET in pulmonary artery endothelial cells]. ApoB100 Remodeling and Stiffened Cholesteryl Ester Core Raise LDL Aggregation in Familial Hypercholesterolemia Patients. Development and application of an LC-MS/MS method for the combined quantification of oxysterols and bile acids. Novel Loci for Triglyceride / High-density Lipoprotein Cholesterol Ratio Longitudinal Change among Subjects without Type 2 Diabetes. Corrigendum to: Protein kinase C activation stabilizes LDL receptor mRNA via the JNK pathway in HepG2 cells [Journal of Lipid Research 50/3 (2008) 386-397].
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