Kazuyo Moro:建立关系对于获得研究速度和机会至关重要。

IF 10.6 1区 医学 Q1 IMMUNOLOGY Journal of Experimental Medicine Pub Date : 2025-03-03 Epub Date: 2024-12-04 DOI:10.1084/jem.20242176
Montserrat Cols
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引用次数: 0

摘要

Kazuyo Moro教授担任RIKEN IMS先天免疫系统实验室和大阪大学医学研究生院的双重任命。她的实验室对2型先天淋巴样细胞(ILC2)进行多方面的研究,从ILC2的分化、激活、抑制和转录控制机制,以及基础研究和药物发现。Moro教授的研究旨在为过敏、纤维化、代谢性疾病等相关免疫疾病建立新的模型和治疗方法。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

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Kazuyo Moro: Building relationships is essential for gaining both speed and opportunities in research.

Professor Kazuyo Moro holds dual appointments as a team leader for the Laboratory for Innate Immune Systems at RIKEN IMS as well as Osaka University Graduate School of Medicine. Her lab conducts multifaceted research on type 2 innate lymphoid cells (ILC2), from ILC2 differentiation, activation, suppression, and transcriptional control mechanisms, as well as basic research and drug discovery. The research from Prof. Moro lab aims to build new models and therapies for related immune diseases such as allergies, fibrosis, and metabolic diseases.

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来源期刊
CiteScore
26.60
自引率
1.30%
发文量
189
审稿时长
3-8 weeks
期刊介绍: Since its establishment in 1896, the Journal of Experimental Medicine (JEM) has steadfastly pursued the publication of enduring and exceptional studies in medical biology. In an era where numerous publishing groups are introducing specialized journals, we recognize the importance of offering a distinguished platform for studies that seamlessly integrate various disciplines within the pathogenesis field. Our unique editorial system, driven by a commitment to exceptional author service, involves two collaborative groups of editors: professional editors with robust scientific backgrounds and full-time practicing scientists. Each paper undergoes evaluation by at least one editor from both groups before external review. Weekly editorial meetings facilitate comprehensive discussions on papers, incorporating external referee comments, and ensure swift decisions without unnecessary demands for extensive revisions. Encompassing human studies and diverse in vivo experimental models of human disease, our focus within medical biology spans genetics, inflammation, immunity, infectious disease, cancer, vascular biology, metabolic disorders, neuroscience, and stem cell biology. We eagerly welcome reports ranging from atomic-level analyses to clinical interventions that unveil new mechanistic insights.
期刊最新文献
Complement-regulated homeostatic proliferation controls memory B cell longevity and repertoire composition. CNOT3 supports ILC2 differentiation and function by destabilizing Tbx21 and Rorc transcripts. Regulatory T cells sabotage anti-tumor γδ T cells by creating IL-2-deficient environments. Reprogramming endogenous NK circuits by highly efficient nonviral genome editing. AID and TET2 cooperate to demethylate Irf4 for plasma cell fate in germinal center B cells.
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