妊娠期间过高的高胆固醇血症是子痫前期内皮功能障碍的危险因素。

IF 4.7 2区 医学 Q1 NEUROSCIENCES Journal of Physiology-London Pub Date : 2024-12-26 DOI:10.1113/JP285943
Amanda A de Oliveira, Floor Spaans, Christy-Lynn M Cooke, Sandra T Davidge
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引用次数: 0

摘要

妊娠引起母体心血管系统的显著变化,血管内皮对妊娠的适应不足导致妊娠并发症的发生,如先兆子痫。先兆子痫不仅是孕产妇发病和死亡的主要原因,也是晚年心血管疾病发展的重要危险因素。然而,子痫前期病理生理学的具体机制,以及晚年对心血管疾病易感性增加的机制,尚未得到充分的表征。在这篇综述中,我们讨论了过量的妊娠特异性血脂异常血症,特别是高胆固醇血症,是先兆子痫发展的一个重要危险因素。我们进一步概述了妊娠期间(在子痫前期)过高胆固醇水平引起的内皮功能障碍的潜在机制(即氧化低密度脂蛋白受体1和toll样受体4),并讨论了妊娠合并子痫前期对后期产妇血管健康的总体影响。确定过量的妊娠期特异性血脂异常/高胆固醇血症影响妊娠期及以后产妇内皮健康的机制,将为妇女易感人群提供诊断和开发靶向治疗的机会,旨在最终减轻心血管疾病的社会负担。
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Excessive hypercholesterolaemia during pregnancy as a risk factor for endothelial dysfunction in pre-eclampsia.

Pregnancy induces significant changes in the maternal cardiovascular system, and insufficient vascular endothelial adaptations to pregnancy contribute to the development of pregnancy complications such as pre-eclampsia. Pre-eclampsia is not only a major cause of maternal morbidity and mortality, but also a significant risk factor for the development of later-life cardiovascular disease. However, the specific mechanisms underlying the pathophysiology of pre-eclampsia, as well as the mechanisms for an increased susceptibility to cardiovascular disease later in life, are not fully characterized. In this review, we discuss the concept that excessive pregnancy-specific dyslipidaemia, particularly hypercholesterolaemia, is a significant risk factor for the development of pre-eclampsia. We further outline novel potential mechanisms (i.e. oxidized low-density lipoprotein receptor 1 and toll-like receptor 4) underlying endothelial dysfunction induced by excessively high cholesterol levels during pregnancy (in the context of pre-eclampsia), in addition to discussing the overall implications of having had a pregnancy complicated by pre-eclampsia on later-life maternal vascular health. Determining the mechanisms by which excessive, pregnancy-specific dyslipidaemia/hypercholesterolaemia impact maternal endothelial health in pregnancy, and later in life, will create a window of opportunity to diagnose and develop targeted therapy for a susceptible population of women, aiming to ultimately reduce the societal burden of cardiovascular disease.

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来源期刊
Journal of Physiology-London
Journal of Physiology-London 医学-神经科学
CiteScore
9.70
自引率
7.30%
发文量
817
审稿时长
2 months
期刊介绍: The Journal of Physiology publishes full-length original Research Papers and Techniques for Physiology, which are short papers aimed at disseminating new techniques for physiological research. Articles solicited by the Editorial Board include Perspectives, Symposium Reports and Topical Reviews, which highlight areas of special physiological interest. CrossTalk articles are short editorial-style invited articles framing a debate between experts in the field on controversial topics. Letters to the Editor and Journal Club articles are also published. All categories of papers are subjected to peer reivew. The Journal of Physiology welcomes submitted research papers in all areas of physiology. Authors should present original work that illustrates new physiological principles or mechanisms. Papers on work at the molecular level, at the level of the cell membrane, single cells, tissues or organs and on systems physiology are all acceptable. Theoretical papers and papers that use computational models to further our understanding of physiological processes will be considered if based on experimentally derived data and if the hypothesis advanced is directly amenable to experimental testing. While emphasis is on human and mammalian physiology, work on lower vertebrate or invertebrate preparations may be suitable if it furthers the understanding of the functioning of other organisms including mammals.
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