STX-721,一种共价、有效、高度突变选择性的EGFR/HER2 Exon20插入抑制剂,用于治疗非小细胞肺癌

IF 6.8 1区 医学 Q1 CHEMISTRY, MEDICINAL Journal of Medicinal Chemistry Pub Date : 2025-01-17 DOI:10.1021/acs.jmedchem.4c02377
Benjamin C. Milgram, Deanna R. Borrelli, Natasja Brooijmans, Jack A. Henderson, Brendan J. Hilbert, Michael R. Huff, Takahiro Ito, Erica L. Jackson, Philip Jonsson, Brendon Ladd, Erin L. O’Hearn, Raymond A. Pagliarini, Simon A. Roberts, Sébastien Ronseaux, Darrin D. Stuart, Weixue Wang, Angel Guzman-Perez
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引用次数: 0

摘要

在L858R和ex19del表皮生长因子受体(EGFR)突变之后,ex20ins突变是非小细胞肺癌(NSCLC)中第三类最常见的驱动突变。不幸的是,第一代、第二代和第三代EGFR酪氨酸激酶抑制剂(TKIs)通常对ex20ins患者无效,因为与野生型EGFR相比,突变活性和选择性不足,导致剂量限制性毒性。虽然近年来在识别有效的EGFR ex20ins突变抑制剂方面取得了重大进展,但突变型与野生型EGFR的选择性仍然是一个重大挑战。STX-721(53)是一种有效的、不可逆的EGFR/HER2 ex20ins突变体抑制剂,在体外和体内均表现出出色的突变型与野生型的选择性。STX-721目前正处于EGFR/HER2 ex20in驱动的NSCLC的1/2期临床试验中。
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Discovery of STX-721, a Covalent, Potent, and Highly Mutant-Selective EGFR/HER2 Exon20 Insertion Inhibitor for the Treatment of Non-Small Cell Lung Cancer
After L858R and ex19del epidermal growth factor receptor (EGFR) mutations, ex20ins mutations are the third most common class of driver-mutations in non-small cell lung cancer (NSCLC). Unfortunately, first-, second-, and third-generation EGFR tyrosine kinase inhibitors (TKIs) are generally ineffective for ex20ins patients due to insufficient mutant activity and selectivity over wild-type EGFR, leading to dose-limiting toxicities. While significant advances in recent years have been made toward identifying potent EGFR ex20ins mutant inhibitors, mutant vs wild-type EGFR selectivity remains a significant challenge. STX-721 (53) is a potent, irreversible inhibitor of the majority of EGFR/HER2 ex20ins mutants and demonstrates excellent mutant vs wild-type selectivity both in vitro and in vivo. STX-721 is currently in phase 1/2 clinical trials for EGFR/HER2 ex20ins-driven NSCLC.
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来源期刊
Journal of Medicinal Chemistry
Journal of Medicinal Chemistry 医学-医药化学
CiteScore
4.00
自引率
11.00%
发文量
804
审稿时长
1.9 months
期刊介绍: The Journal of Medicinal Chemistry is a prestigious biweekly peer-reviewed publication that focuses on the multifaceted field of medicinal chemistry. Since its inception in 1959 as the Journal of Medicinal and Pharmaceutical Chemistry, it has evolved to become a cornerstone in the dissemination of research findings related to the design, synthesis, and development of therapeutic agents. The Journal of Medicinal Chemistry is recognized for its significant impact in the scientific community, as evidenced by its 2022 impact factor of 7.3. This metric reflects the journal's influence and the importance of its content in shaping the future of drug discovery and development. The journal serves as a vital resource for chemists, pharmacologists, and other researchers interested in the molecular mechanisms of drug action and the optimization of therapeutic compounds.
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