骨关节炎中下垂铁特征基因的转录组学分析和实验验证。

IF 2.4 4区 医学 Q2 RHEUMATOLOGY International Journal of Rheumatic Diseases Pub Date : 2025-01-01 DOI:10.1111/1756-185X.70083
Zhen-Yu Gao, Guo-Qing Yan, Linchong Su, San-Shan He, Jiao-E Sheng, Qing-Chao Wu, Xia Huang, Yu-Fang Dai
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引用次数: 0

摘要

骨关节炎是一种全身性疾病,主要损害关节软骨,也影响滑膜、韧带和骨组织。涉及的关键机制是软骨细胞死亡和细胞外基质降解。本研究旨在鉴定与铁下垂相关的差异表达基因(DEGs),并探讨其在骨关节炎发展中的作用。我们采用转录组数据分析、基因富集分析、蛋白-蛋白相互作用网络构建、动物模型实验、免疫细胞浸润分析等多种方法。我们对GSE114007数据集的检查发现了2614个基因,其中1300个基因上调,1314个基因下调。由此,我们确定了8个与铁中毒相关的DEGs (FRGs-DEGs)。功能富集分析表明,这些基因对细胞迁移和组织重塑具有重要意义。它们特别参与HIF-1和PPAR信号通路。此外,我们的免疫细胞浸润分析显示骨关节炎样品中M0和M2巨噬细胞增加,而嗜酸性粒细胞和记忆B细胞水平明显降低。受试者工作特征曲线分析发现GJA1、TIMP金属肽酶抑制剂1 (TIMP1)和DPP4是骨关节炎诊断的潜在生物标志物,曲线下面积分别为0.91、0.85和0.83。此外,在骨关节炎大鼠模型中进行的RT-qPCR验证证实了TIMP1的上调,支持我们的生物信息学结果。总之,我们的研究确定了关键的FRGs-DEGs及其在骨关节炎中的潜在作用。这项研究为该病的分子机制提供了新的见解,并为临床干预提供了创新的治疗靶点。未来的研究应旨在包括更大的患者队列和临床验证,以提高这些发现的适用性。
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Transcriptomic Analysis and Experimental Verification of Ferroptosis Signature Genes in Osteoarthritis.

Osteoarthritis is a systemic disease that primarily damages articular cartilage and also affects the synovium, ligaments, and bone tissues. The key mechanisms involved are chondrocyte death and degradation of the extracellular matrix. This study aims to identify differentially expressed genes (DEGs) associated with ferroptosis and investigate their roles in the development of osteoarthritis. We used several methods, such as transcriptomic data analysis, gene enrichment analysis, protein-protein interaction network construction, animal model experiments, and immune cell infiltration analysis. Our examination of the GSE114007 dataset uncovered 2614 DEGs, including 1300 that were upregulated and 1314 that were downregulated. From these, we identified eight ferroptosis-related DEGs (FRGs-DEGs). Functional enrichment analysis showed that these genes are significant for cellular migration and tissue remodeling. They are particularly involved in the HIF-1 and PPAR signaling pathways. Additionally, our immune cell infiltration analysis indicated an increase in M0 and M2 macrophages in osteoarthritis samples, while levels of eosinophils and memory B cells were notably decreased. The receiver operating characteristic curve analysis identified GJA1, TIMP Metallopeptidase Inhibitor 1 (TIMP1), and DPP4 as potential biomarkers for osteoarthritis diagnosis, with area under the curve of 0.91, 0.85, and 0.83, respectively. Moreover, RT-qPCR validation in an osteoarthritis rat model confirmed the upregulation of TIMP1, supporting our bioinformatics results. In summary, our study identifies key FRGs-DEGs and their potential roles in osteoarthritis. This research provides new insights into the disease's molecular mechanisms and suggests innovative therapeutic targets for clinical intervention. Future research should aim to include larger patient cohorts and clinical validation to improve the applicability of these findings.

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来源期刊
CiteScore
3.70
自引率
4.00%
发文量
362
审稿时长
1 months
期刊介绍: The International Journal of Rheumatic Diseases (formerly APLAR Journal of Rheumatology) is the official journal of the Asia Pacific League of Associations for Rheumatology. The Journal accepts original articles on clinical or experimental research pertinent to the rheumatic diseases, work on connective tissue diseases and other immune and allergic disorders. The acceptance criteria for all papers are the quality and originality of the research and its significance to our readership. Except where otherwise stated, manuscripts are peer reviewed by two anonymous reviewers and the Editor.
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