【撤回】Tan IIA通过MDM4‑IAP3信号通路抑制H1299细胞活力。

IF 3.4 3区 医学 Q2 MEDICINE, RESEARCH & EXPERIMENTAL Molecular medicine reports Pub Date : 2025-03-01 Epub Date: 2025-01-17 DOI:10.3892/mmr.2025.13435
Yukun Zu, Jianning Wang, Wei Ping, Wei Sun
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引用次数: 0

摘要

在上述论文发表后,一位关心的读者提请编辑注意,2386页图1C和D中所示的某些western blotting数据与另一家研究机构的不同作者所写的其他两篇文章中以不同形式出现的数据惊人地相似,这些文章在提交给《分子医学报告》之前已经在其他地方发表过。此外,图6A和图C中的一些数据惊人地相似,也表明该图中的数据是错误组装的。鉴于上述数据显然已经在之前发表过,《分子医学报告》编辑决定从期刊上撤回这篇论文。作者被要求对这些担忧作出解释,但编辑部没有收到答复。对于由此给读者带来的不便,本刊编辑深表歉意。[分子医学报告]17:2384‑2392,2018;DOI: 10.3892 / mmr.2017.8152]。
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[Retracted] Tan IIA inhibits H1299 cell viability through the MDM4‑IAP3 signaling pathway.

Following the publication of the above paper, it was drawn to the Editors' attention by a concerned reader that certain of the western blotting data shown in Fig. 1C and D on p. 2386 were strikingly similar to data appearing in different form in a pair of other articles written by different authors at a different research institute that had already been published elsewhere prior to the submission of this paper to Molecular Medicine Reports. Moreover, some of the data featured in Fig. 6A and C were strikingly similar, also suggesting that the data in this figure had been misassembled. In view of the fact that the abovementioned data had already apparently been published previously, the Editor of Molecular Medicine Reports has decided that this paper should be retracted from the Journal. The authors were asked for an explanation to account for these concerns, but the Editorial Office did not receive a reply. The Editor apologizes to the readership for any inconvenience caused. [Molecular Medicine Reports 17: 2384‑2392, 2018; DOI: 10.3892/mmr.2017.8152].

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来源期刊
Molecular medicine reports
Molecular medicine reports 医学-病理学
CiteScore
7.60
自引率
0.00%
发文量
321
审稿时长
1.5 months
期刊介绍: Molecular Medicine Reports is a monthly, peer-reviewed journal available in print and online, that includes studies devoted to molecular medicine, underscoring aspects including pharmacology, pathology, genetics, neurosciences, infectious diseases, molecular cardiology and molecular surgery. In vitro and in vivo studies of experimental model systems pertaining to the mechanisms of a variety of diseases offer researchers the necessary tools and knowledge with which to aid the diagnosis and treatment of human diseases.
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