磷接力反馈回路控制刚地弓形虫的出口与细胞内发育。

IF 7.7 1区 生物学 Q1 CELL BIOLOGY Cell reports Pub Date : 2025-02-25 Epub Date: 2025-02-03 DOI:10.1016/j.celrep.2025.115260
Ja E Claywell, Yong Fu, L David Sibley
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引用次数: 0

摘要

细胞内寄生虫刚地弓形虫在运动侵袭和静止细胞内复制形式之间交替,但这些转变是如何调节的尚不清楚。一个包括蛋白激酶G (PKG)和钙依赖性PKs (CDPKs)的正反馈回路控制着弓形虫的运动、侵袭和出口,而PKA异构体c1 (PKAc1)抵消了这一途径。侵袭后不久,PKAc1被腺苷酸环化酶产生的环AMP (cAMP)激活,导致PKG/CDPK途径受到抑制。PKAc1进一步激活磷酸二酯酶2,磷酸二酯酶2选择性消耗cAMP,形成负反馈回路,导致PKAc1瞬间激活。环GMP (cGMP)对钙的扰动表明PKAc1作用于鸟苷酸环化酶和钙释放之间的靶标。PKG/CDPKs的联合激活和PKAc1的抑制,由一个短暂的负反馈回路控制,确保了寄生虫对激活运动所需的环境信号做出反应,同时也确保了长期稳定的细胞内生长。
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Phospho-relay feedback loops control egress vs. intracellular development in Toxoplasma gondii.

The intracellular parasite Toxoplasma gondii alternates between a motile invasive and a quiescent intracellular replicative form, yet how these transitions are regulated is unknown. A positive feedback loop involving protein kinase G (PKG) and calcium-dependent PKs (CDPKs) controls motility, invasion, and egress by Toxoplasma gondii, while PKA isoform c1 (PKAc1) counteracts this pathway. Shortly after invasion, PKAc1 is activated by cyclic AMP (cAMP) produced by adenylate cyclases, leading to the suppression of the PKG/CDPK pathway. PKAc1 further activates phosphodiesterase 2, which selectively consumes cAMP, thus forming a negative feedback loop, causing transient activation of PKAc1. Perturbation of cyclic GMP (cGMP) vs. calcium demonstrates that PKAc1 acts on targets between guanylate cyclase and calcium release. The combined activation of PKG/CDPKs and inhibition by PKAc1, controlled by a transient negative feedback loop, ensures that the parasite is responsive to environmental signals needed to activate motility while also ensuring periods of long-term stable intracellular growth.

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来源期刊
Cell reports
Cell reports CELL BIOLOGY-
CiteScore
13.80
自引率
1.10%
发文量
1305
审稿时长
77 days
期刊介绍: Cell Reports publishes high-quality research across the life sciences and focuses on new biological insight as its primary criterion for publication. The journal offers three primary article types: Reports, which are shorter single-point articles, research articles, which are longer and provide deeper mechanistic insights, and resources, which highlight significant technical advances or major informational datasets that contribute to biological advances. Reviews covering recent literature in emerging and active fields are also accepted. The Cell Reports Portfolio includes gold open-access journals that cover life, medical, and physical sciences, and its mission is to make cutting-edge research and methodologies available to a wide readership. The journal's professional in-house editors work closely with authors, reviewers, and the scientific advisory board, which consists of current and future leaders in their respective fields. The advisory board guides the scope, content, and quality of the journal, but editorial decisions are independently made by the in-house scientific editors of Cell Reports.
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