E3连接酶TRIM8通过k48关联的多泛素化作用靶向MYOF降解抑制肺癌转移。

IF 9.6 1区 生物学 Q1 CELL BIOLOGY Cell Death & Disease Pub Date : 2025-02-11 DOI:10.1038/s41419-025-07421-6
Chi-Hsuan Wei, Chia-Wei Weng, Chih-Ying Wu, Hsuan-Yu Chen, Ya-Hsuan Chang, Gee-Chen Chang, Jeremy J W Chen
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引用次数: 0

摘要

泛素化是一种翻译后修饰,通过泛素-蛋白酶体系统调节肿瘤进展相关蛋白,使E3连接酶成为潜在的抗肿瘤靶点。在这里,我们报道TRIM8, TRIM家族成员和E3连接酶,可以作为非小细胞肺癌(NSCLC)的肿瘤抑制因子。功能增益和功能缺失实验均显示TRIM8抑制NSCLC细胞的增殖、集落形成、迁移和侵袭。异种移植瘤模型实验表明,TRIM8的表达在体内抑制肿瘤转移。此外,TRIM8的低表达与台湾和GEO肺癌队列中较差的总生存率相关。TRIM8在肺癌细胞中的过表达降低了MYOF的表达,并在TRIM8过表达的细胞中恢复MYOF拯救的细胞迁移。TRIM8靶向MYOF进行k48相关的泛素化,促进蛋白酶体介导的降解,随后抑制MMPs的细胞外分泌。我们的研究结果为TRIM8对肺癌进展的贡献提供了新的见解,表明TRIM8是一种新的生物标志物和新的肺癌治疗靶点。
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E3 ligase TRIM8 suppresses lung cancer metastasis by targeting MYOF degradation through K48-linked polyubiquitination.

Ubiquitination is a posttranslational modification that regulates tumour progression-associated proteins through the ubiquitin‒proteasome system, making E3 ligases potential antitumour targets. Here, we report that TRIM8, a member of the TRIM family and an E3 ligase, can act as a tumour suppressor in non-small cell lung cancer (NSCLC). Both gain- and loss-of-function experiments revealed that TRIM8 inhibits the proliferation, colony formation, migration and invasion of NSCLC cells. Experiments with a xenograft model showed that TRIM8 expression suppresses tumour metastasis in vivo. Moreover, low expression of TRIM8 was associated with poor overall survival in both the Taiwanese and GEO lung cancer cohorts. TRIM8 overexpression in lung cancer cells reduced MYOF expression, and restoring MYOF rescued cell migration in TRIM8-overexpressing cells. TRIM8 targeted MYOF for K48-linked ubiquitination, facilitating proteasome-mediated degradation and subsequently suppressing the extracellular secretion of MMPs. Our results provide new insights into the contribution of TRIM8 to lung cancer progression, suggesting that TRIM8 is a new biomarker and a novel therapeutic target for lung cancer.

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来源期刊
Cell Death & Disease
Cell Death & Disease CELL BIOLOGY-
CiteScore
15.10
自引率
2.20%
发文量
935
审稿时长
2 months
期刊介绍: Brought to readers by the editorial team of Cell Death & Differentiation, Cell Death & Disease is an online peer-reviewed journal specializing in translational cell death research. It covers a wide range of topics in experimental and internal medicine, including cancer, immunity, neuroscience, and now cancer metabolism. Cell Death & Disease seeks to encompass the breadth of translational implications of cell death, and topics of particular concentration will include, but are not limited to, the following: Experimental medicine Cancer Immunity Internal medicine Neuroscience Cancer metabolism
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