产生额颞叶痴呆和阿尔茨海默病的非人灵长类动物模型的实验基础。

IF 3.1 3区 医学 Q2 NEUROSCIENCES Journal of Alzheimer's Disease Pub Date : 2025-04-01 Epub Date: 2025-03-02 DOI:10.1177/13872877251321116
Takahiro Morito, Naoto Watamura, Hiroki Sasaguri, Taisuke Tomita, Makoto Higuchi, Hideyuki Okano, Erika Sasaki, Takaomi C Saido
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引用次数: 0

摘要

据我们所知,目前还没有报道描述与17号染色体(FTDP-17)相关的非人类灵长类动物(NHP)额颞叶痴呆伴帕金森病模型不依赖于过表达范式。基于我们最近成功地生成单个人MAPT敲入FTDP-17小鼠模型,我们描述了生成敲入FTDP-17的绒猴模型的实验基础。此外,成功生成缺乏阿尔茨海默病(AD)外显子9的突变型PSEN1敲入狨猴模型(PSEN1-Δ9)表明,通过结合这些模型,我们将能够加速重建AD的两个主要病理特征,即淀粉样斑块和神经原纤维缠结。
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Experimental basis for generating nonhuman primate models of frontotemporal dementia and Alzheimer's disease.

To our knowledge, no reports have described nonhuman primate (NHP) models of frontotemporal dementia with Parkinsonism linked to chromosome 17 (FTDP-17) that do not depend on an overexpression paradigm. Based on our recent success in generating single human MAPT knock-in mouse models of FTDP-17, we describe the experimental basis for generating knock-in marmoset models of FTDP-17. In addition, successful generation of mutant PSEN1 knock-in marmoset models lacking exon 9 (PSEN1-Δ9) of Alzheimer's disease (AD) indicates that we will be able to reconstitute two major pathological features of AD, i.e., amyloid plaques and neurofibrillary tangles, in an accelerated manner by combining these models.

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来源期刊
Journal of Alzheimer's Disease
Journal of Alzheimer's Disease 医学-神经科学
CiteScore
6.40
自引率
7.50%
发文量
1327
审稿时长
2 months
期刊介绍: The Journal of Alzheimer''s Disease (JAD) is an international multidisciplinary journal to facilitate progress in understanding the etiology, pathogenesis, epidemiology, genetics, behavior, treatment and psychology of Alzheimer''s disease. The journal publishes research reports, reviews, short communications, hypotheses, ethics reviews, book reviews, and letters-to-the-editor. The journal is dedicated to providing an open forum for original research that will expedite our fundamental understanding of Alzheimer''s disease.
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