{"title":"慢性消化不良的胃十二指肠运动。2. 餐后运动)。","authors":"S Sielaff","doi":"","DOIUrl":null,"url":null,"abstract":"<p><p>After ingestion of a solid test meal the postprandial motor activity in 17 dyspeptic patients and 12 healthy controls were examined. In all individuals the gastric emptying was measured by scintigraphy. - The antral pressure activity after food intake was delayed in dyspepsia and showed a distinct reduction with time (antral hypomotility). In contrast the postprandial duodenal motility was increased significantly (duodenal hyperdyskinesia). All 6 dyspeptic patients with prolonged gastric emptying had gastroduodenal manometric abnormalities. - Our results suggest that in chronic dyspepsia the interdigestive and postprandial motility is often disturbed. The delayed gastric emptying occurs because of impaired antral peristalsis and/or increase of duodenal resistance.</p>","PeriodicalId":11189,"journal":{"name":"Deutsche Zeitschrift fur Verdauungs- und Stoffwechselkrankheiten","volume":"48 3-4","pages":"162-72"},"PeriodicalIF":0.0000,"publicationDate":"1988-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"[Gastroduodenal motility in chronic dyspepsia. 2. Postprandial motility].\",\"authors\":\"S Sielaff\",\"doi\":\"\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><p>After ingestion of a solid test meal the postprandial motor activity in 17 dyspeptic patients and 12 healthy controls were examined. In all individuals the gastric emptying was measured by scintigraphy. - The antral pressure activity after food intake was delayed in dyspepsia and showed a distinct reduction with time (antral hypomotility). In contrast the postprandial duodenal motility was increased significantly (duodenal hyperdyskinesia). All 6 dyspeptic patients with prolonged gastric emptying had gastroduodenal manometric abnormalities. - Our results suggest that in chronic dyspepsia the interdigestive and postprandial motility is often disturbed. The delayed gastric emptying occurs because of impaired antral peristalsis and/or increase of duodenal resistance.</p>\",\"PeriodicalId\":11189,\"journal\":{\"name\":\"Deutsche Zeitschrift fur Verdauungs- und Stoffwechselkrankheiten\",\"volume\":\"48 3-4\",\"pages\":\"162-72\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"1988-01-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Deutsche Zeitschrift fur Verdauungs- und Stoffwechselkrankheiten\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Deutsche Zeitschrift fur Verdauungs- und Stoffwechselkrankheiten","FirstCategoryId":"1085","ListUrlMain":"","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
[Gastroduodenal motility in chronic dyspepsia. 2. Postprandial motility].
After ingestion of a solid test meal the postprandial motor activity in 17 dyspeptic patients and 12 healthy controls were examined. In all individuals the gastric emptying was measured by scintigraphy. - The antral pressure activity after food intake was delayed in dyspepsia and showed a distinct reduction with time (antral hypomotility). In contrast the postprandial duodenal motility was increased significantly (duodenal hyperdyskinesia). All 6 dyspeptic patients with prolonged gastric emptying had gastroduodenal manometric abnormalities. - Our results suggest that in chronic dyspepsia the interdigestive and postprandial motility is often disturbed. The delayed gastric emptying occurs because of impaired antral peristalsis and/or increase of duodenal resistance.