一氧化碳中毒致急性肺水肿。预后的影响。

A Durocher, F Saulnier, D Dubois, F Fourrier, C Chopin, F Wattel
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摘要

从1980年7月至1982年7月,我们的重症监护病房收治了285例一氧化碳中毒患者。18例患者出现肺水肿(pe)。昏迷2、3、4级时多发P.E. (p < 10(-3))。Knauss提出的急性生理评分(生理测量加权)在P.E.患者中较高(p < 10(-2))。然而,死亡从来不是因为体育;昏迷54例,死亡5例;死亡与神经系统恶化有关。神经系统后遗症与P.E.的发生无关,而与高压氧治疗的延迟有关。这些数据表明,一氧化碳中毒的预后与神经系统状况有关,在正确治疗和早期高压氧治疗的情况下,不受P.E.的影响。
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[Acute pulmonary edema in carbon monoxide poisoning. Prognostic effect].

285 patients affected by carbon monoxide poisoning were admitted in our intensive care unit over a period of two years (from july 1980 to july 1982). 18 patients had a pulmonary edema (P.E.). The occurrence of P.E. was more frequent when coma was grade 2, 3 or 4 (p less than 10(-3]. The acute physiologic score (weighting of physiologic measurements) as proposed by Knauss is higher in patients with P.E. (p less than 10(-2]. However death is never due to P.E.; about 54 patients with coma, 5 died; death is related to neurologic aggravation. Neurologic sequelae are not related to the occurrence of P.E. but to a delay in hyperbaric oxygen therapy. These date show that prognosis of carbon monoxide poisoning is related to neurological status and is not influenced by the occurrence of P.E. when correctly treated and when hyperbaric oxygen therapy is early realized.

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