{"title":"[2例代谢不良低钾血症性麻痹伴补钾期神经肌肉兴奋性增高的病因分析]。","authors":"M R Caccia","doi":"","DOIUrl":null,"url":null,"abstract":"<p><p>Increased neuromuscular excitability with varying clinical and EMG features were also observed during KCl administration in both cases. The findings are discussed on the light of the membrane ionic gradients current theory.</p>","PeriodicalId":21409,"journal":{"name":"Rivista di patologia nervosa e mentale","volume":"104 3","pages":"97-104"},"PeriodicalIF":0.0000,"publicationDate":"1983-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"[Etiopathogenetic considerations in 2 cases of dysmetabolic hypokalemic paralysis with signs of increased neuromuscular excitability during potassium repletion].\",\"authors\":\"M R Caccia\",\"doi\":\"\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><p>Increased neuromuscular excitability with varying clinical and EMG features were also observed during KCl administration in both cases. The findings are discussed on the light of the membrane ionic gradients current theory.</p>\",\"PeriodicalId\":21409,\"journal\":{\"name\":\"Rivista di patologia nervosa e mentale\",\"volume\":\"104 3\",\"pages\":\"97-104\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"1983-05-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Rivista di patologia nervosa e mentale\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Rivista di patologia nervosa e mentale","FirstCategoryId":"1085","ListUrlMain":"","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
[Etiopathogenetic considerations in 2 cases of dysmetabolic hypokalemic paralysis with signs of increased neuromuscular excitability during potassium repletion].
Increased neuromuscular excitability with varying clinical and EMG features were also observed during KCl administration in both cases. The findings are discussed on the light of the membrane ionic gradients current theory.
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