[大鼠胎儿红细胞生成的调控:肝红细胞生成能力不足时脾和骨髓的作用]。

M D Nagel, J M Felix, J Nagel
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引用次数: 0

摘要

在大鼠的子宫内生命结束时,肝红细胞的进化是由皮质类固醇控制的,实验上可以通过对怀孕动物施加反复的应激来过早地剥夺胎儿的大部分肝红细胞生成组织。这种实验诱导的肝红细胞缺乏导致骨髓中预期的红细胞活性。脾切除术对这类胎儿的治疗表明,脾在足月前产生循环无核红细胞中不起主要作用。
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[Regulation of erythropoiesis in the rat fetus: role of spleen and bone marrow in case of deficiency of hepatic erythropoietic activity].

At the end of intra-uterine life in the rat, hepatic erythron evolution is governed by corticosteroids and it is experimentally possible to prematurely deprive the foetus of a large part of its hepatic erythropoietic tissue by inflicting repeated stress in the pregnant animal. This experimentally induced deficiency of the hepatic erythron leads to an anticipated erythroid activity in the bone marrow. Splenectomy in such fetuses has shown that the spleen does not play a major role in producing circulating anucleated erythroid cells before term.

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