[急性髓性白血病患者颅内粒细胞肉瘤]。

No to shinkei = Brain and nerve Pub Date : 2006-09-01
Harue Obara, Shinjitsu Nishimura, Nakamasa Hayashi, Yoshihiro Numagami, Tomoo Inoue, Kohmei Kubo, Mitsuomi Kaimori, Michiharu Nishijima
{"title":"[急性髓性白血病患者颅内粒细胞肉瘤]。","authors":"Harue Obara,&nbsp;Shinjitsu Nishimura,&nbsp;Nakamasa Hayashi,&nbsp;Yoshihiro Numagami,&nbsp;Tomoo Inoue,&nbsp;Kohmei Kubo,&nbsp;Mitsuomi Kaimori,&nbsp;Michiharu Nishijima","doi":"","DOIUrl":null,"url":null,"abstract":"<p><p>Granulocytic sarcoma (GS) is extramedullary tumor composed of immature leukemic cells. GS is presenting usually as a complication during the course of hematologic neoplasm, such as acute myeloblastic leukemia as well as myeloproliferative and myelodysplastic syndrome. The tumor was also called chroloma based on the green color of the tumorous mass. Central nervous system manifestations of GS are extremely rare. We report a 41-year-old man with acute leukemia type M7, who developed GS in the right occipital lobe after complete remission was achieved. Operative findings revealed the color of the hard tumor was greenish, which suggested the tumor was chroloma. Histological findings showed the tumor was GS. The majority of reported cases of GS in acute myeloid leukemia were M2 FAB classification and have chromosome translocation. Our patient was M7 FAB classification, not have specific chromosome translocation. GS occurrence in AML: M7 patient was extremely rare. This is the first case report of AML: M7 with GS in the central nervous system.</p>","PeriodicalId":19163,"journal":{"name":"No to shinkei = Brain and nerve","volume":"58 9","pages":"797-801"},"PeriodicalIF":0.0000,"publicationDate":"2006-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"[Intracranial granulocytic sarcoma in a patient with acute myeloid leukemia].\",\"authors\":\"Harue Obara,&nbsp;Shinjitsu Nishimura,&nbsp;Nakamasa Hayashi,&nbsp;Yoshihiro Numagami,&nbsp;Tomoo Inoue,&nbsp;Kohmei Kubo,&nbsp;Mitsuomi Kaimori,&nbsp;Michiharu Nishijima\",\"doi\":\"\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><p>Granulocytic sarcoma (GS) is extramedullary tumor composed of immature leukemic cells. GS is presenting usually as a complication during the course of hematologic neoplasm, such as acute myeloblastic leukemia as well as myeloproliferative and myelodysplastic syndrome. The tumor was also called chroloma based on the green color of the tumorous mass. Central nervous system manifestations of GS are extremely rare. We report a 41-year-old man with acute leukemia type M7, who developed GS in the right occipital lobe after complete remission was achieved. Operative findings revealed the color of the hard tumor was greenish, which suggested the tumor was chroloma. Histological findings showed the tumor was GS. The majority of reported cases of GS in acute myeloid leukemia were M2 FAB classification and have chromosome translocation. Our patient was M7 FAB classification, not have specific chromosome translocation. GS occurrence in AML: M7 patient was extremely rare. This is the first case report of AML: M7 with GS in the central nervous system.</p>\",\"PeriodicalId\":19163,\"journal\":{\"name\":\"No to shinkei = Brain and nerve\",\"volume\":\"58 9\",\"pages\":\"797-801\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2006-09-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"No to shinkei = Brain and nerve\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"No to shinkei = Brain and nerve","FirstCategoryId":"1085","ListUrlMain":"","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 0

摘要

粒细胞肉瘤(GS)是由未成熟的白血病细胞组成的髓外肿瘤。GS通常表现为血液肿瘤病程中的并发症,如急性髓母细胞白血病以及骨髓增生性和骨髓增生异常综合征。根据肿瘤肿块的绿色,该肿瘤也被称为色素瘤。GS的中枢神经系统表现极为罕见。我们报告一个41岁的男性急性白血病M7型,谁在完全缓解后发展GS在右侧枕叶。手术发现硬瘤呈绿色,提示为色肌瘤。组织学表现为GS型肿瘤。报告的急性髓系白血病GS多为M2 FAB型,并有染色体易位。本例患者为M7 FAB型,无特异性染色体易位。在AML: M7患者中,GS的发生极为罕见。这是首例AML: M7伴中枢神经系统GS的病例报道。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
分享 分享
微信好友 朋友圈 QQ好友 复制链接
本刊更多论文
[Intracranial granulocytic sarcoma in a patient with acute myeloid leukemia].

Granulocytic sarcoma (GS) is extramedullary tumor composed of immature leukemic cells. GS is presenting usually as a complication during the course of hematologic neoplasm, such as acute myeloblastic leukemia as well as myeloproliferative and myelodysplastic syndrome. The tumor was also called chroloma based on the green color of the tumorous mass. Central nervous system manifestations of GS are extremely rare. We report a 41-year-old man with acute leukemia type M7, who developed GS in the right occipital lobe after complete remission was achieved. Operative findings revealed the color of the hard tumor was greenish, which suggested the tumor was chroloma. Histological findings showed the tumor was GS. The majority of reported cases of GS in acute myeloid leukemia were M2 FAB classification and have chromosome translocation. Our patient was M7 FAB classification, not have specific chromosome translocation. GS occurrence in AML: M7 patient was extremely rare. This is the first case report of AML: M7 with GS in the central nervous system.

求助全文
通过发布文献求助,成功后即可免费获取论文全文。 去求助
来源期刊
自引率
0.00%
发文量
0
期刊最新文献
[Metabolic myopathy]. [Myasthenia gravis]. [Myasthenia gravis]. [Genetic changes in JC virus possibly associated with progressive multifocal leukoencephalopathy]. [Neuropathology of progressive multifocal leukoencephalopathy].
×
引用
GB/T 7714-2015
复制
MLA
复制
APA
复制
导出至
BibTeX EndNote RefMan NoteFirst NoteExpress
×
×
提示
您的信息不完整,为了账户安全,请先补充。
现在去补充
×
提示
您因"违规操作"
具体请查看互助需知
我知道了
×
提示
现在去查看 取消
×
提示
确定
0
微信
客服QQ
Book学术公众号 扫码关注我们
反馈
×
意见反馈
请填写您的意见或建议
请填写您的手机或邮箱
已复制链接
已复制链接
快去分享给好友吧!
我知道了
×
扫码分享
扫码分享
Book学术官方微信
Book学术文献互助
Book学术文献互助群
群 号:481959085
Book学术
文献互助 智能选刊 最新文献 互助须知 联系我们:info@booksci.cn
Book学术提供免费学术资源搜索服务,方便国内外学者检索中英文文献。致力于提供最便捷和优质的服务体验。
Copyright © 2023 Book学术 All rights reserved.
ghs 京公网安备 11010802042870号 京ICP备2023020795号-1