胃旁路手术会改变体重设定值吗?

International journal of obesity supplements Pub Date : 2016-12-01 Epub Date: 2016-11-16 DOI:10.1038/ijosup.2016.9
Z Hao, M B Mumphrey, C D Morrison, H Münzberg, J Ye, H R Berthoud
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引用次数: 15

摘要

成年期相对稳定的体重归因于大脑中存在的一种稳态调节机制,该机制利用身体的反馈来控制能量的摄入和消耗。这种机制保证,如果被设计的扰动上升或下降,体重将返回到扰动前的水平,定义为防御水平或设定点。节食后体重反弹很常见的事实表明,肥胖的人会保持较高的体重水平。因此,体重的设定值是灵活的,可能是由遗传、表观遗传和环境因素的复杂相互作用决定的。与节食不同,减肥手术在持续抑制食物摄入和体重方面做得更好,对潜在机制的深入研究已经开始。虽然对于Roux-en-Y胃旁路手术(RYGB)的这种持久影响的一种解释是由于侵入性手术造成的简单的物理限制,但更令人兴奋的解释是手术在生理上重新编程了体重防御机制。在这篇非系统综述中,我们提出了来自我们自己和其他研究的行为证据,表明RYGB和袖式胃切除术后防御体重降低。在这些手术后,如果一段时间内食物过量或不足,啮齿动物会恢复到它们喜欢的较低体重,并且在合成代谢阶段急剧增加食物摄入量的能力有力地反驳了身体限制假说。然而,潜在的机制仍然不清楚。虽然机制涉及中枢瘦素和黑素皮质素信号通路,但其他外周信号,如肠道激素及其神经效应通路可能也有贡献。未来在人类和啮齿类动物中使用靶向和非靶向“组学”技术,以及在啮齿类动物中使用现代的、基因靶向的神经操纵技术的研究将是必要的。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

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Does gastric bypass surgery change body weight set point?

The relatively stable body weight during adulthood is attributed to a homeostatic regulatory mechanism residing in the brain which uses feedback from the body to control energy intake and expenditure. This mechanism guarantees that if perturbed up or down by design, body weight will return to pre-perturbation levels, defined as the defended level or set point. The fact that weight re-gain is common after dieting suggests that obese subjects defend a higher level of body weight. Thus, the set point for body weight is flexible and likely determined by the complex interaction of genetic, epigenetic and environmental factors. Unlike dieting, bariatric surgery does a much better job in producing sustained suppression of food intake and body weight, and an intensive search for the underlying mechanisms has started. Although one explanation for this lasting effect of particularly Roux-en-Y gastric bypass surgery (RYGB) is simple physical restriction due to the invasive surgery, a more exciting explanation is that the surgery physiologically reprograms the body weight defense mechanism. In this non-systematic review, we present behavioral evidence from our own and other studies that defended body weight is lowered after RYGB and sleeve gastrectomy. After these surgeries, rodents return to their preferred lower body weight if over- or underfed for a period of time, and the ability to drastically increase food intake during the anabolic phase strongly argues against the physical restriction hypothesis. However, the underlying mechanisms remain obscure. Although the mechanism involves central leptin and melanocortin signaling pathways, other peripheral signals such as gut hormones and their neural effector pathways likely contribute. Future research using both targeted and non-targeted 'omics' techniques in both humans and rodents as well as modern, genetically targeted, neuronal manipulation techniques in rodents will be necessary.

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