ME7瘙痒病感染小鼠滤泡辅助性T细胞反应延长。

IF 1.9 3区 生物学 Q4 BIOCHEMISTRY & MOLECULAR BIOLOGY Prion Pub Date : 2018-03-04 Epub Date: 2018-04-20 DOI:10.1080/19336896.2018.1458573
Soochan Kim, Sinsuk Han, Taehyun Kim, Jeehoon Nam, Yong-Sun Kim, Eun-Kyoung Choi, Mi-Yeon Kim
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引用次数: 2

摘要

我们之前报道过,小鼠脑内接种小鼠适应的痒病菌株ME7后,由于CCL19和CCL21的表达降低,脾脏T区明显减少。此外,me7感染的脾脏生发中心的滤泡树突状细胞网络比未感染的脾脏大。作为该研究的延伸,我们开始确定ME7感染如何影响小鼠脾脏结构和滤泡辅助T (Tfh)细胞反应。在本研究中,小鼠腹腔注射ME7疫苗的脑匀浆,并在接种后50、130和200天对小鼠的脾脏进行分析,并与未感染小鼠进行比较。结果表明,ME7感染小鼠的Tfh细胞反应增加,并一直维持到终末期朊病毒病。尽管CD4 T细胞在白浆中减少,但它们在生发中心增加,并表达更高水平的tfh相关基因,如Bcl6、Il21、Cxcr5、Icos和Pdcd1。此外,me7感染小鼠脾脏CD4记忆T细胞数量增加。这些数据表明,尽管ME7感染导致脾白髓结构受损,但CD4记忆T细胞增加,Tfh细胞反应需要并延长,以帮助致病性朊蛋白在生发中心的复制和积累。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

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Prolonged follicular helper T cell responses in ME7 scrapie-infected mice.

We previously reported that mice intracerebrally inoculated with the mouse-adapted scrapie strain ME7 have markedly diminished T zones in the spleen due to the decreased expression of CCL19 and CCL21. In addition, follicular dendritic cell networks in germinal centers were larger in ME7-infected spleens compared to uninfected spleens. As an extension of that study, we set out to determine how ME7 infection affects spleen structure and follicular helper T (Tfh) cell responses in mice. For this study, mice were intraperitoneally inoculated with brain homogenate of the ME7 inoculum and spleens were analyzed 50, 130, and 200 days after inoculation and compared with those from uninfected mice. The result showed that ME7- infected mice had increased Tfh cell responses which were maintained until end-stage prion disease. Although CD4 T cells decreased in white pulps, they increased in germinal centers, and expressed higher levels of the Tfh-related genes, such as Bcl6, Il21, Cxcr5, Icos, and Pdcd1. In addition, ME7-infected spleens had increased numbers of CD4 memory T cells. These data indicate that although ME7 infection led to impaired splenic white pulp structure, CD4 memory T cells were increased and Tfh cell responses were required and prolonged to provide help for the replication and accumulation of pathogenic prion protein in germinal centers.

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来源期刊
Prion
Prion 生物-生化与分子生物学
CiteScore
5.20
自引率
4.30%
发文量
13
审稿时长
6-12 weeks
期刊介绍: Prion is the first international peer-reviewed open access journal to focus exclusively on protein folding and misfolding, protein assembly disorders, protein-based and structural inheritance. The goal is to foster communication and rapid exchange of information through timely publication of important results using traditional as well as electronic formats. The overriding criteria for publication in Prion are originality, scientific merit and general interest.
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