脊柱背支注射和射频神经松解术治疗骨质疏松性胸腰椎压缩性骨折所致腰痛。

Linqiu Zhou, Jeffrey Zhou
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摘要

目的:骨质疏松性胸腰椎压缩性骨折引起的腰痛是一种常见的衰弱性疾病。本研究的目的是确定脊髓背支注射和射频神经松解术对减轻这种疾病患者疼痛的准确性和有效性。方法:对46例骨质疏松性胸腰椎压缩性骨折所致腰痛患者进行回顾性分析。所有患者均接受脊髓背支注射混合Sensorcaine (Fresenius Kabi,美国)和Depo-Medrol (Pfizer,美国)治疗。在一些患者中,在初始注射消退后需要进一步的射频神经松解治疗。结果:在46例患者中,45例(97.7%)患者注射后腰痛立即减轻≥50%。在最初的注射消退后,18名患者仍然没有疼痛,27名患者需要射频神经松解术。随访期60 ~ 1440天(平均335天)。腰痛强度由治疗前的7.09±0.84(数值疼痛评分0-10)降至注射后的1.39±1.51,最后一次就诊时为0.96±1.36。结论:脊柱背支注射联合射频神经松解术是治疗骨质疏松性胸腰椎压缩性骨折所致腰痛的有效、准确的治疗方法。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

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Spinal Dorsal Rami Injection and Radiofrequency Neurolysis for Low Back Pain Caused by Osteoporosis-Induced Thoracolumbar Vertebral Compression Fractures.

Objective: Low back pain caused by osteoporosisinduced thoracolumbar vertebral compression fractures is a common debilitating disorder. The aims of this study were to determine the accuracy and efficacy of spinal dorsal ramus injection and radiofrequency neurolysis for pain reduction in patients with this condition.

Methods: This study was a retrospective chart review of 46 patients with low back pain caused by osteoporosis-induced thoracolumbar vertebral compression fractures. All patients had been treated with spinal dorsal ramus injection with mixed Sensorcaine (Fresenius Kabi, USA) and Depo-Medrol (Pfizer, USA). In some patients further treatment with radiofrequency neurolysis had been required after the initial injection wore off.

Results: Out of a total of 46 patients, 45 (97.7%) had ≥ 50% reduction in low back pain immediately after injection. After the initial injection wore off, 18 patients remained pain free and 27 required radiofrequency neurolysis. The follow-up period ranged from 60 to 1,440 days (mean 335 days). The intensity of low back pain decreased from 7.09 ± 0.84 (numerical pain scale of 0-10) before treatment to 1.39 ± 1.51 immediately after the injection, and to 0.96 ± 1.36 at the last office visit.

Conclusion: Spinal dorsal ramus injection and radiofrequency neurolysis are effective and accurate therapies for low back pain caused by osteoporosis-induced thoracolumbar vertebral compression fractures.

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