脂肪细胞瘦素在阴道毛滴虫诱导前列腺细胞增殖中的信号作用。

IF 1.4 4区 医学 Q3 PARASITOLOGY Korean Journal of Parasitology Pub Date : 2021-06-01 Epub Date: 2021-06-21 DOI:10.3347/kjp.2021.59.3.235
Jung-Hyun Kim, Ik-Hwan Han, Su-Jin Shin, Sung-Yul Park, Hyo-Yeoung Chung, Jae-Sook Ryu
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引用次数: 6

摘要

瘦素是一种主要由脂肪细胞产生的脂肪因子,据报道在前列腺癌中过量产生。然而,尚不清楚它是否会刺激前列腺细胞的增殖。在这项研究中,我们研究了感染阴道毛滴虫的良性前列腺增生上皮细胞(BPH-1细胞)是否通过瘦素信号通路诱导前列腺细胞增殖。为了研究脂肪细胞瘦素与炎症上皮细胞之间的串扰对前列腺细胞增殖的影响,我们将脂肪细胞3T3-L1细胞置于感染阴道t细胞的BPH-1细胞条件培养基(T. vaginalis-conditioned medium, TCM)中培养,然后鉴定脂肪细胞条件培养基(ATCM)对前列腺细胞的增殖有促进作用。BPH-1细胞与活阴道t细胞孵育后释放促炎细胞因子,这些细胞的条件培养基引起脂肪细胞的迁移。当前列腺基质细胞和BPH-1细胞与含有瘦素的脂肪细胞条件培养基孵育时,它们的生长速度增加,瘦素受体(OBR)和JAK2/STAT3、Notch和survivin等信号分子的表达也增加。此外,阻断OBR可减少ATCM的增殖和瘦素信号分子的表达。总之,我们的研究结果表明,感染阴道t细胞的炎症BPH-1细胞通过瘦素- obr信号诱导前列腺细胞增殖。因此,生殖道绦虫可能通过前列腺炎症释放的脂肪细胞瘦素导致前列腺肥大。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

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Signaling Role of Adipocyte Leptin in Prostate Cell Proliferation Induced by Trichomonas vaginalis.

Leptin is a type of adipokine mainly produced by adipocytes and reported to be overproduced in prostate cancer. However, it is not known whether it stimulates the proliferation of prostate cells. In this study, we investigated whether benign prostatic hyperplasia epithelial cells (BPH-1 cells) infected with Trichomonas vaginalis induced the proliferation of prostate cells via a leptin signaling pathway. To investigate the effect of crosstalk between adipocyte leptin and inflamed epithelial cell in proliferation of prostate cells, adipocytes 3T3-L1 cells were incubated in conditioned medium of BPH-1 cells infected with T. vaginalis (T. vaginalis-conditioned medium, TCM), and then the adipocyte-conditioned medium (ATCM) was identified to cause proliferation of prostate cells. BPH-1 cells incubated with live T. vaginalis released pro-inflammatory cytokines, and conditioned medium of these cells caused migration of adipocytes. When prostate stromal cells and BPH-1 cells were incubated with adipocyte conditioned medium containing leptin, their growth rates increased as did expression of the leptin receptor (known as OBR) and signaling molecules such as JAK2/STAT3, Notch and survivin. Moreover, blocking the OBR reduced this proliferation and the expression of leptin signaling molecules in response to ATCM. In conclusion, our findings show that inflamed BPH-1 cells infected with T. vaginalis induce the proliferation of prostate cells through leptin-OBR signaling. Therefore, it is likely that T. vaginalis contributes to prostate enlargement in BPH via adipocyte leptin released as a result of inflammation of the prostate.

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来源期刊
CiteScore
2.80
自引率
0.00%
发文量
48
审稿时长
1 months
期刊介绍: The Korean Journal of Parasitology is the official journal paperless, on-line publication after Vol. 53, 2015 of The Korean Society for Parasitology and Tropical Medicine. Abbreviated title is ‘Korean J Parasitol’. It was launched in 1963. It contains original articles, case reports, brief communications, reviews or mini-reviews, book reviews, and letters to the editor on parasites of humans and animals, vectors, host-parasite relationships, zoonoses, and tropical medicine. It is published bimonthly in February, April, June, August, October, and December each year. Supplement numbers are at times published. All of the manuscripts are peer-reviewed.
期刊最新文献
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