MiR-18a-3p 通过抑制 PDP1 改善骨关节炎的软骨基质重塑并抑制炎症。

IF 4.6 Q2 MATERIALS SCIENCE, BIOMATERIALS ACS Applied Bio Materials Pub Date : 2022-02-11 DOI:10.1186/s12576-022-00827-3
Xiaoguang Feng, Jiajun Lu, Yixiong Wu, Haiyun Xu
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引用次数: 4

摘要

骨关节炎(OA)是一种以滑膜炎症为特征的退行性疾病。据报道,OA 患者膝关节前交叉韧带中的 miR-18a-3p 出现下调。本研究探讨了 miR-18a-3p 在 OA 中的具体功能和机制。研究人员使用 10 ng/ml IL-1β 处理 ATDC5 细胞,建立了 OA 体外模型,并对 Wistar 大鼠进行了内侧半月板失稳手术,建立了 OA 体内大鼠模型。RT-qPCR显示,在IL-1β刺激的ATDC5细胞中,miR-18a-3p被下调。通过酶联免疫吸附试验(ELISA)和免疫印迹法(Western blotting)显示,miR-18a-3p 的过表达抑制了炎症细胞因子的分泌和基质金属蛋白酶的浓度。荧光素酶报告实验检测了 miR-18a-3p 与丙酮酸脱氢酶磷酸化酶催化亚基 1(PDP1)的结合关系。MiR-18a-3p 靶向 PDP1 并负向调节 PDP1 的表达。挽救实验的结果显示,PDP1 的上调保留了 miR-18a-3p 过表达对 IL-1β 刺激的 ATDC5 细胞中炎症细胞因子和基质金属蛋白酶水平的抑制作用。用 H&E 染色法观察 Wistar 大鼠膝关节滑膜组织的病理变化。Safranin O-快绿素/红霉素用于染色膝关节软骨样本。MiR-18a-3p的过表达抑制了体内OA的进展。总之,miR-18a-3p 通过靶向 PDP1 改善软骨基质重塑并抑制 OA 中的炎症。
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MiR-18a-3p improves cartilage matrix remodeling and inhibits inflammation in osteoarthritis by suppressing PDP1.

Osteoarthritis (OA) is a degenerative disease characterized by synovial inflammation. MiR-18a-3p was reported to be downregulated in knee anterior cruciate ligament of OA patients. In the present study, the specific functions and mechanism of miR-18a-3p in OA were explored. An in vitro model of OA was established using 10 ng/ml IL-1β to treat ATDC5 cells, and medial meniscus instability surgery was performed on Wistar rats to establish in vivo rat model of OA. RT-qPCR revealed that miR-18a-3p was downregulated in IL-1β-stimulated ATDC5 cells. MiR-18a-3p overexpression inhibited secretion of inflammatory cytokines and concentration of matrix metalloproteinases, as shown by ELISA and western blotting. The binding relation between miR-18a-3p and pyruvate dehydrogenase phosphatase catalytic subunit 1 (PDP1) was detected by luciferase reporter assays. MiR-18a-3p targeted PDP1 and negatively regulated PDP1 expression. Results of rescue assays revealed that PDP1 upregulation reserved the suppressive effect of miR-18a-3p overexpression on levels of inflammatory cytokines and matrix metalloproteinases in IL-1β-stimulated ATDC5 cells. H&E staining was used to observe pathological changes of synovial tissues in the knee joint of Wistar rats. Safranin O-fast green/hematoxylin was used to stain cartilage samples of knee joints. MiR-18a-3p overexpression suppressed OA progression in vivo. Overall, miR-18a-3p improves cartilage matrix remodeling and suppresses inflammation in OA by targeting PDP1.

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来源期刊
ACS Applied Bio Materials
ACS Applied Bio Materials Chemistry-Chemistry (all)
CiteScore
9.40
自引率
2.10%
发文量
464
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