范可尼贫血途径受损导致人血管肉瘤DNA低甲基化。

IF 4.3 3区 生物学 Human Cell Pub Date : 2022-09-01 Epub Date: 2022-07-11 DOI:10.1007/s13577-022-00736-y
Kangning Zhu, Suofeng Sun, Fengxia Guo, Lan Gao
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引用次数: 1

摘要

血管肉瘤(AS)是一种罕见的软组织肉瘤,治疗方案差,预后差。异常DNA甲基化模式已被确定与不同血管肉瘤亚型具有一定的临床相关性。然而,其深层机制尚不清楚。在本研究中,我们研究了36例伴有或不伴有慢性淋巴水肿的AS,并报道了DNA损伤是导致DNA甲基化异常的重要因素。此外,我们确定范可尼贫血(FA)通路受损导致AS伴慢性淋巴水肿的严重DNA损伤。我们还观察到激活的FANCD2可以促进DNMT1在基因组DNA上的募集。我们的研究揭示了FA通路对DNA甲基化的新调控机制,有助于进一步了解AS的发病机制,并为AS的治疗提供潜在的治疗靶点。
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Impaired Fanconi anemia pathway causes DNA hypomethylation in human angiosarcomas.

Angiosarcomas (AS) is a rare soft tissue sarcomas with poor treatment options and a dismal prognosis. The abnormal DNA methylation pattern has been determined as the certain clinical relevance with different angiosarcoma subtypes. However, the profound mechanism is not clear. In present study, we studied thirty-six AS with or without chronic lymphedema, and reported that DNA damage was an important factor causing DNA methylation abnormality. Furthermore, we determined that the impaired Fanconi anemia (FA) pathway contributed to severe DNA damage in AS with chronic lymphedema. We also observed that the activated FANCD2 could facilitate DNMT1 recruitment on genomic DNA. Our study uncovers a novel regulatory mechanism of FA pathway on DNA methylation, and is a benefit to advanced understanding the pathogenesis of AS, as well as providing the potential therapeutic targets for AS treatment.

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来源期刊
Human Cell
Human Cell 生物-细胞生物学
CiteScore
6.60
自引率
2.30%
发文量
176
期刊介绍: Human Cell is the official English-language journal of the Japan Human Cell Society. The journal serves as a forum for international research on all aspects of the human cell, encompassing not only cell biology but also pathology, cytology, and oncology, including clinical oncology. Embryonic stem cells derived from animals, regenerative medicine using animal cells, and experimental animal models with implications for human diseases are covered as well. Submissions in any of the following categories will be considered: Research Articles, Cell Lines, Rapid Communications, Reviews, and Letters to the Editor. A brief clinical case report focusing on cellular responses to pathological insults in human studies may also be submitted as a Letter to the Editor in a concise and short format. Not only basic scientists but also gynecologists, oncologists, and other clinical scientists are welcome to submit work expressing new ideas or research using human cells.
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