格林-巴-罗综合征伴肌酸激酶升高的弛缓性四肢瘫1例并文献复习

Q4 Nursing Journal of Neurocritical Care Pub Date : 2021-11-15 DOI:10.18700/jnc.210011

B. Mahesh, A. Pathak, R. Chaurasia, Anand B Kumar, V. K. Singh

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引用次数: 0

摘要

格林-巴利综合征（GBS）是一种急性暴发性多神经根神经病，具有自身免疫性，以急性弛缓性麻痹为特征，伴有或不伴有感觉异常。GBS主要分为脱髓鞘型和轴突型。三种常见的亚型是急性炎症性脱髓鞘多发性神经根病变、急性运动轴索神经病变（AMAN）和急性运动和感觉轴索神经损伤（AMSAN）[1]。肌酸激酶（CK）催化肌酸和三磷酸腺苷分解为磷酸肌酸，并伴有肌酸激酶升高的格林-巴利综合征偏瘫：一例病例报告及文献复习

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Flaccid quadriparesis with raised creatine kinase in Guillain-Barré syndrome: a case report with review of literature

Guillain-Barré syndrome (GBS) is an acute and fulminant polyradiculoneuropathy that is autoimmune in nature and characterized by acute flaccid paralysis, with or without sensory abnormalities. GBS is predominantly classified as demyelinating and axonal types. The three common subtypes are acute inflammatory demyelinating polyradiculoneuropathy, acute motor axonal neuropathy (AMAN), and acute motor and sensory axonal neuropathy (AMSAN) [1]. Creatine kinase (CK) catalyzes the breakdown of creatine and adenosine triphosphate into phosphocreatine and Flaccid quadriparesis with raised creatine kinase in Guillain-Barré syndrome: a case report with review of literature

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