腹腔镜胃束带置入术后假性贲门失弛缓症1例

Venkata Kollimarla, Akhila Rachakonda, J. Myers, Steven Knox, S. Thompson
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引用次数: 0

摘要

背景:腹腔镜可调节胃束带(LAGB)是治疗肥胖的常见方法。LAGB的一个潜在并发症是假性贲门失弛缓症(一种食道运动障碍)。在选定的个体中,LAGB可能会产生高的流出阻力,导致食管远端的高压环境,从而导致进行性虚弱和扩张。假性贲门失弛缓症的治疗取决于逆转根本原因。病例描述:一名64岁女性,患有病态肥胖[体重指数(BMI)41kg/m2]和裂孔疝,接受腹腔镜胃束带插入术。作为手术的一部分,用永久性编织缝线进行了裂孔修复。术后,患者体重减轻了30公斤,但开始注意到反流和吞咽困难。一年后,腹腔镜带被摘除,但这并没有缓解她的症状。内窥镜检查显示食道异常扩张,充满液体。在接下来的24个月里,患者接受了四次内镜扩张,但收效甚微。在第四次扩张时,患者出现吸入性肺炎,导致住院时间过长。最后,通过腹腔镜下切除前裂孔修复并移除(LAGB)囊膜,解决了根本原因。不幸的是,在接下来的12个月里,患者的症状没有得到改善,于是进行了一次困难的腹腔镜心肌切开术。患者随后病情好转,能够耐受正常饮食。结论:本病例报告强调了在处理假性贲门失弛缓症时逆转所有潜在潜在病因的关键性(即,切除LAGB和纤维化包膜;拆除先前的裂孔修复和/或胃底折叠术)。同样,最重要的是,本病例报告提醒读者,对于有严重反流和吞咽困难症状的患者,在内窥镜检查期间必须保护气道,以防止误吸。
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Pseudoachalasia following insertion of a laparoscopic gastric band: a case report
Background: Laparoscopic adjustable gastric banding (LAGB) is a common procedure to treat obesity. A potential complication of LAGB is pseudoachalasia (an esophageal motility disorder). In select individuals, a LAGB may create high outflow resistance, leading to a high-pressure environment in the distal esophagus, which then leads to progressive weakness and dilatation. Treatment of pseudoachalasia hinges on reversing the underlying cause. Case Description: A 64-year-old female, with morbid obesity [body mass index (BMI) 41 kg/m 2 ] and a hiatus hernia, underwent laparoscopic insertion of a gastric band. As part of her procedure, a hiatal repair was performed with permanent braided sutures. Post-operatively, the patient lost 30 kg, however began to notice regurgitation and dysphagia. The laparoscopic band was removed a year later, but this did not alleviate her symptoms. Endoscopy showed an abnormal, dilated, fluid-filled esophagus. The patient underwent four endoscopic dilations over the next 24 months, with minimal benefit. On the fourth dilatation, the patient aspirated and developed aspiration pneumonia, resulting in a lengthy admission. Finally, the underlying cause was addressed with a laparoscopic takedown of the anterior hiatal repair and removal of the capsule (from the LAGB). Unfortunately, the patient’s symptoms failed to improve over the next 12 months, and a difficult laparoscopic cardiomyotomy was performed. The patient subsequently improved and was then able to tolerate a normal diet. Conclusions: This case report highlights the critical nature of reversing all potential underlying causes when dealing with pseudoachalasia (i.e., removal of the LAGB and fibrotic capsule; takedown of a prior hiatal repair and/or fundoplication). As well, and of utmost importance, this case report reminds the reader that in a patient with severe symptoms of regurgitation and dysphagia, the airway must be protected during endoscopy to prevent aspiration.
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