Sri Mulyani, Sutia Budi, Indra Cahyono, Khairiman Khairiman
{"title":"天然饲料中维生素C生物胶囊化对遮目鱼幼鱼蛋白质、脂肪、能量及死亡率的影响","authors":"Sri Mulyani, Sutia Budi, Indra Cahyono, Khairiman Khairiman","doi":"10.14710/jkt.v26i2.17969","DOIUrl":null,"url":null,"abstract":"Indications of milkfish larvae problems show lack symptoms of vitamin C in the tissues, which will cause defective bone formation and become an inhibitor factor in growth and mortality. This research aims to investigate the influence of vitamin C bioencapsulation on rotifer and artemia in protein retention, fat, energy, and the cause of death in milkfish larvae (Chanos chanos). The dose of vitamin C used in this study were 0 (control), 50, 100, 150, and 200 mg L-1. The treatment starts from D-1 to D-30 stadia of milkfish larvae, with 30-day maintenance. The density of milkfish larvae utilized is 20 L-1. The protein and fat content of milkfish larvae bodies were measured using a proximate analysis, and the energy content of the feed was measured using a bomb calorimeter. Mortality was observed under the Ministry of Health of Republic Indonesia standard binocular microscope by identifying larvae that died daily in each container. This research shows that vitamin C bioencapsulation in rotifer and artemia affects the retention rate of protein, fat, energy and the cause of milkfish larvae mortality. The dose of vitamin C of 150 and 200 mg L-1 is effective in improving protein, fat, and energy retention, which can minimize the cause of milkfish larvae mortality. The best results are obtained by bioencapsulating Vitamin C on rotifer and artemia with dose of 150 mg L-1.","PeriodicalId":53001,"journal":{"name":"Jurnal Kelautan Tropis","volume":" ","pages":""},"PeriodicalIF":0.0000,"publicationDate":"2023-04-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Effect of Vitamin C Bioencapsulation in Natural Feed on Protein, Fat, Energy, and Mortality of Milkfish Larvae (Chanos chanos)\",\"authors\":\"Sri Mulyani, Sutia Budi, Indra Cahyono, Khairiman Khairiman\",\"doi\":\"10.14710/jkt.v26i2.17969\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Indications of milkfish larvae problems show lack symptoms of vitamin C in the tissues, which will cause defective bone formation and become an inhibitor factor in growth and mortality. This research aims to investigate the influence of vitamin C bioencapsulation on rotifer and artemia in protein retention, fat, energy, and the cause of death in milkfish larvae (Chanos chanos). The dose of vitamin C used in this study were 0 (control), 50, 100, 150, and 200 mg L-1. The treatment starts from D-1 to D-30 stadia of milkfish larvae, with 30-day maintenance. The density of milkfish larvae utilized is 20 L-1. The protein and fat content of milkfish larvae bodies were measured using a proximate analysis, and the energy content of the feed was measured using a bomb calorimeter. Mortality was observed under the Ministry of Health of Republic Indonesia standard binocular microscope by identifying larvae that died daily in each container. This research shows that vitamin C bioencapsulation in rotifer and artemia affects the retention rate of protein, fat, energy and the cause of milkfish larvae mortality. The dose of vitamin C of 150 and 200 mg L-1 is effective in improving protein, fat, and energy retention, which can minimize the cause of milkfish larvae mortality. The best results are obtained by bioencapsulating Vitamin C on rotifer and artemia with dose of 150 mg L-1.\",\"PeriodicalId\":53001,\"journal\":{\"name\":\"Jurnal Kelautan Tropis\",\"volume\":\" \",\"pages\":\"\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2023-04-17\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Jurnal Kelautan Tropis\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.14710/jkt.v26i2.17969\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Jurnal Kelautan Tropis","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.14710/jkt.v26i2.17969","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Effect of Vitamin C Bioencapsulation in Natural Feed on Protein, Fat, Energy, and Mortality of Milkfish Larvae (Chanos chanos)
Indications of milkfish larvae problems show lack symptoms of vitamin C in the tissues, which will cause defective bone formation and become an inhibitor factor in growth and mortality. This research aims to investigate the influence of vitamin C bioencapsulation on rotifer and artemia in protein retention, fat, energy, and the cause of death in milkfish larvae (Chanos chanos). The dose of vitamin C used in this study were 0 (control), 50, 100, 150, and 200 mg L-1. The treatment starts from D-1 to D-30 stadia of milkfish larvae, with 30-day maintenance. The density of milkfish larvae utilized is 20 L-1. The protein and fat content of milkfish larvae bodies were measured using a proximate analysis, and the energy content of the feed was measured using a bomb calorimeter. Mortality was observed under the Ministry of Health of Republic Indonesia standard binocular microscope by identifying larvae that died daily in each container. This research shows that vitamin C bioencapsulation in rotifer and artemia affects the retention rate of protein, fat, energy and the cause of milkfish larvae mortality. The dose of vitamin C of 150 and 200 mg L-1 is effective in improving protein, fat, and energy retention, which can minimize the cause of milkfish larvae mortality. The best results are obtained by bioencapsulating Vitamin C on rotifer and artemia with dose of 150 mg L-1.