Re: Wright H, Fahmy M, Bourke G.副肌作为肘管近端尺神经压迫的可能原因:1例报告。中华手外科杂志,2016,31 (4):559 - 561

G. Georgiev
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(2017), the lack of knowledge of the variant muscle discovered at surgical intervention could be (1) scarcely presented clinical reports, (2) rarity of the reported structure and (3) limited approach during decompression surgery. I would like to add another cause: a limited knowledge about anatomical variations. According to me, when accepting a variant muscle, three important things should be born in mind: (1) location, (2) insertions and (3) function. In the article of Wright et al. (2021) the approach is not limited and gives wide visualization of the muscular variant and the authors could clearly determine it. In my opinion, it could be speculated that the possible cause of ulnar nerve compression in the reported case is accessory slip from the triceps brachii muscle. Accessory slip from triceps brachii, although rare, is clearly described in the work of Swamy et al. (2013). 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引用次数: 1

摘要

我饶有兴趣地读了这篇文章。作者描述了一个案例的副肌作为一个可能的原因尺神经压迫近肘管。然而,我想对报道的副肌做一些简单的评论。作者正确地指出,这块肌肉不具有上睑闭锁肌的解剖特征,在位置上更近端。然而,我认为,当作者接受一种肌肉变异作为神经压迫的可能原因时,他们需要更清楚,应该提出肌肉变异的可能名称或解释它属于哪一种肌肉。正如Georgiev等人(2017)所指出的那样,缺乏对手术干预中发现的变异肌肉的了解可能是:(1)几乎没有临床报道,(2)报道的结构罕见,(3)减压手术时的入路有限。我想补充另一个原因:对解剖学变异的了解有限。据我所知,在接受变异肌肉时,应该记住三个重要的事情:(1)位置,(2)插入,(3)功能。在Wright等人(2021)的文章中,该方法不受限制,并提供了肌肉变体的广泛可视化,作者可以清楚地确定它。在我看来,可以推测,报告的病例中尺神经压迫的可能原因是肱三头肌的副滑脱。来自肱三头肌的辅助滑移,虽然罕见,但在Swamy等人(2013)的工作中有明确描述。此外,Kim等人(2016)也描述了一例肱三头肌额外滑动导致尺神经受压的临床病例。综上所述,笔者认为,在考虑变异性肌肉卡压神经病时,作者应提出其所属肌肉的建议,而不是只报道副肌。解剖学是一门非常古老的科学,对于经验丰富的外科医生来说,精确了解不同的解剖学变异是必不可少的。
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Re: Wright H, Fahmy M, Bourke G. An accessory muscle as the possible cause of ulnar nerve compression proximal to the cubital canal: a case report. J Hand Surg Eur. 2021, 46: 1114–5
I read this article with interest. The authors described a case of an accessory muscle as a possible cause of ulnar nerve compression proximal to the cubital canal. However, I would like to make my modest comments about the reported accessory muscle. The authors correctly point out that this muscle does not have the anatomical features of the anconeus epitrochlearis muscle and was more proximal in location. However, I think that when the authors accept a variant muscle as a possible cause of nerve compression, they need to be clearer and should make a proposal about the possible name of the muscular variation or explain to which muscle it belongs. As pointed out by Georgiev et al. (2017), the lack of knowledge of the variant muscle discovered at surgical intervention could be (1) scarcely presented clinical reports, (2) rarity of the reported structure and (3) limited approach during decompression surgery. I would like to add another cause: a limited knowledge about anatomical variations. According to me, when accepting a variant muscle, three important things should be born in mind: (1) location, (2) insertions and (3) function. In the article of Wright et al. (2021) the approach is not limited and gives wide visualization of the muscular variant and the authors could clearly determine it. In my opinion, it could be speculated that the possible cause of ulnar nerve compression in the reported case is accessory slip from the triceps brachii muscle. Accessory slip from triceps brachii, although rare, is clearly described in the work of Swamy et al. (2013). Moreover, a clinical case of ulnar nerve compression by additional slips of the triceps brachii has also been described by Kim et al. (2016). In conclusion, I think that when considering variant muscle entrapment neuropathy, the authors should make a proposal about the muscle to which it belongs, rather than only report an accessory muscle. Anatomy is a very old science, and precise knowledge of different anatomical variations is mandatory for experienced surgeons.
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