ARB、MRA、saireito联合治疗膜性肾病肾病综合征的缓解诱导疗效

T. Ono, H. Nagai, Noriyuki Suzuki, F. Nogaki
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引用次数: 1

摘要

血管紧张素受体阻滞剂(ARBs)可有效降低尿蛋白。ARB后醛固酮升高被称为醛固酮突破,而矿皮质激素受体拮抗剂(MRAs)对这种现象有用。日本的传统药物“海藤”具有减少蛋白尿的潜力。mra可以抑制甘草诱导的假醛固酮增多症。
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Remission inductive efficacy of the combination of ARB, MRA, and saireito for nephrotic syndrome of membranous nephropathy
Angiotensin II receptor blockers (ARBs) effectively reduce urinary protein. An increase in aldosterone after ARB is termed aldosterone breakthrough and mineralocorticoid receptor antagonists (MRAs) are useful against this phenomenon. The Japanese traditional medicine saireito has the potential to reduce proteinuria. MRAs may suppress licorice‐induced pseudoaldosteronism.
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