{"title":"阿尔茨海默病的影响及其策略","authors":"Khadar Khan","doi":"10.35248/0975-0851.21.S3.001","DOIUrl":null,"url":null,"abstract":"Alzheimer's infection (AD) is the most widely recognized neurodegenerative issue showed by reformist loss of memory followed by irreversible dementia [1]. Promotion neurodegeneration is described by the deficiency of neurons went before by cell layer and cytoskeleton harm because of a complex of atomic pathways that at long last lead to development of amyloid-β (Aβ) plaques and neurofibrillary tangles in the mind tissue.","PeriodicalId":15184,"journal":{"name":"Journal of Bioequivalence & Bioavailability","volume":"144 1","pages":"1-2"},"PeriodicalIF":0.0000,"publicationDate":"2021-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Alzheimer’s Disease Implications and its Strategies\",\"authors\":\"Khadar Khan\",\"doi\":\"10.35248/0975-0851.21.S3.001\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Alzheimer's infection (AD) is the most widely recognized neurodegenerative issue showed by reformist loss of memory followed by irreversible dementia [1]. Promotion neurodegeneration is described by the deficiency of neurons went before by cell layer and cytoskeleton harm because of a complex of atomic pathways that at long last lead to development of amyloid-β (Aβ) plaques and neurofibrillary tangles in the mind tissue.\",\"PeriodicalId\":15184,\"journal\":{\"name\":\"Journal of Bioequivalence & Bioavailability\",\"volume\":\"144 1\",\"pages\":\"1-2\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2021-01-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Journal of Bioequivalence & Bioavailability\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.35248/0975-0851.21.S3.001\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of Bioequivalence & Bioavailability","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.35248/0975-0851.21.S3.001","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 0
摘要
阿尔茨海默病感染(Alzheimer's infection, AD)是公认最广泛的神经退行性疾病，表现为改革派记忆丧失，随后出现不可逆痴呆[1]。促进性神经退行性变被描述为细胞层和细胞骨架损伤之前的神经元缺乏，这是由于复杂的原子途径最终导致脑组织中淀粉样蛋白-β (a β)斑块和神经原纤维缠结的发展。
Alzheimer’s Disease Implications and its Strategies
Alzheimer's infection (AD) is the most widely recognized neurodegenerative issue showed by reformist loss of memory followed by irreversible dementia [1]. Promotion neurodegeneration is described by the deficiency of neurons went before by cell layer and cytoskeleton harm because of a complex of atomic pathways that at long last lead to development of amyloid-β (Aβ) plaques and neurofibrillary tangles in the mind tissue.
京公网安备 11010802042870号
京ICP备2023020795号-1
分享
求助内容：
应助结果提醒方式：
扫码关注我们
