RhoC GTPase过表达调节乳腺细胞血管生成因子的诱导。

IF 6.3 2区 医学 Q1 ONCOLOGY Neoplasia Pub Date : 2000-09-01 DOI:10.1038/SJ.NEO.7900115
K. Golen, Zhi-fen Wu, Xiao Tan Qiao, Liwei Bao, S. Merajver
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引用次数: 170
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RhoC GTPase overexpression modulates induction of angiogenic factors in breast cells.
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来源期刊
Neoplasia
Neoplasia ONCOLOGY-
自引率
2.10%
发文量
82
期刊介绍: Neoplasia publishes the results of novel investigations in all areas of oncology research. The title Neoplasia was chosen to convey the journal’s breadth, which encompasses the traditional disciplines of cancer research as well as emerging fields and interdisciplinary investigations. Neoplasia is interested in studies describing new molecular and genetic findings relating to the neoplastic phenotype and in laboratory and clinical studies demonstrating creative applications of advances in the basic sciences to risk assessment, prognostic indications, detection, diagnosis, and treatment. In addition to regular Research Reports, Neoplasia also publishes Reviews and Meeting Reports. Neoplasia is committed to ensuring a thorough, fair, and rapid review and publication schedule to further its mission of serving both the scientific and clinical communities by disseminating important data and ideas in cancer research.
期刊最新文献
DCZ0014, a novel compound in the therapy of diffuse large B-cell lymphoma via the B cell receptor signaling pathway. Corrigendum to "Hodgkin Lymphoma Cell Lines Are Characterized by a Specific miRNA Expression Profile." Neoplasia 2009, Feb;11(2):167-176. The dual PI3K/mTOR inhibitor NVP-BEZ235 is a potent inhibitor of ATM- and DNA-PKCs-mediated DNA damage responses. Sensitization of pancreatic cancer stem cells to gemcitabine by Chk1 inhibition. Astrocytes upregulate survival genes in tumor cells and induce protection from chemotherapy.
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