模拟心肌梗死的心脏结节病。

Kyoko Oyama-Suzuki, Kenji Fukushima, Ryuta Egi, Shintaro Nakano, Toshihiro Muramatsu
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引用次数: 0

摘要

一位58岁无症状的男性心电图异常被转介到我们的机构进行心脏检查。他的心电图显示双束传导阻滞,超声心动图显示心尖壁运动异常。应力-静息心肌灌注显像(MPI)显示前壁明显缺损,静息时部分重分布。他被怀疑患有前路心肌梗死(MI)并接受了心导管插入术。然而,冠状动脉造影(CAG)未显示明显的冠状动脉粥样硬化。行心血管磁共振成像(CMR)评价心肌梗死程度。晚期钆增强(LGE)显示心外膜和心肌中部多部位明显LGE,包括前室间隔、外侧、下壁和右心室基底交界处,这强烈提示纤维化是由心脏结节病(CS)引起的。MPI显示前壁心肌灌注缺损,显示纤维化为非典型发现,与前壁心肌梗死相似。
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Cardiac Sarcoidosis Mimicking Anterior Myocardial Infarction.

A 58-year-old asymptomatic man with electrocardiogram (ECG) abnormality was referred to our institution for cardiac exams. His ECG showed a bifascicular block, and the echocardiography demonstrated a wall motion abnormality in apex. Stress-rest myocardial perfusion imaging (MPI) showed a significant defect in anterior wall with partial redistribution at rest. He was suspected of having an anterior myocardial infarction (MI) and underwent cardiac catheterization. However, coronary angiography (CAG) revealed no significant coronary atherosclerosis. Cardiovascular magnetic resonance imaging (CMR) was performed to evaluate the extent of myocardial infarction. Late gadolinium enhancement (LGE) demonstrated a significant epicardial and midmyocardial LGE in the multiple site including anterior, anteroseptal, lateral, inferior wall, and basal right ventricle junction, which strongly indicated that the fibrosis was due to cardiac sarcoidosis (CS).A myocardial perfusion defect in anterior wall shown in the MPI revealed the fibrosis as an atypical finding mimicking anterior MI.

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