Corrigendum to "Rac1/PAK1 signaling contributes to bone cancer pain by Regulation dendritic spine remodeling in rats".

IF 2.8 3区医学 Q2 NEUROSCIENCES Molecular Pain Pub Date : 2023-01-01 DOI:10.1177/17448069231182501

引用次数: 0

查看原文

微信好友朋友圈 QQ好友复制链接

本刊更多论文

“Rac1/PAK1信号通过调节大鼠树突棘重塑而导致骨癌症疼痛”更正。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

求助全文

约1分钟内获得全文去求助

来源期刊

Molecular Pain 医学-神经科学

CiteScore

5.60

自引率

3.00%

发文量

审稿时长

6-12 weeks

期刊介绍： Molecular Pain is a peer-reviewed, open access journal that considers manuscripts in pain research at the cellular, subcellular and molecular levels. Molecular Pain provides a forum for molecular pain scientists to communicate their research findings in a targeted manner to others in this important and growing field.

期刊最新文献

Neural Adaptation of the Reward System in Primary Dysmenorrhea. Rapid cleavage of IL-1β in DRG neurons produces tissue injury-induced pain hypersensitivity. Analyzing Substance Levels and Pain Perception in Painless Labor: The Impact of Spinal Epidural Analgesia. Assessment of orofacial nociceptive behaviors of mice with the sheltering tube method: Oxaliplatin-induced mechanical and cold allodynia in orofacial regions. Upregulation of KDM6B in the anterior cingulate cortex contributes to neonatal maternal deprivation-induced chronic visceral pain in mice.