Minimally invasive tunneling of a de-epithelialized connective tissue graft to improve gingival phenotype of lingual recession defects: A case report

IF 0.9 Q3 DENTISTRY, ORAL SURGERY & MEDICINE Clinical Advances in Periodontics Pub Date : 2022-10-21 DOI:10.1002/cap.10230
Yoonah Danskin, Stephanie Chu, Trevor Simmonds
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Abstract

Background

This case report describes a minimally invasive technique to increase the functional resistance of mandibular anterior lingual recession defects to inflammation. There are only a few case reports that describe the soft tissue augmentation of lingual gingival recession, of which none describe a tunneling technique without coronal advancement of the flap to treat a long span of multiple recession defects. Soft tissue augmentation of lingual recession defects is challenging due to the proximity to the tongue, frenum, vital structures, pre-existing thin phenotype, and limited access during surgery.

Methods and Results

A 30-year-old male was referred for the treatment of gingival recession on the lingual surfaces of teeth #22–27, with a diagnosis of recession type 2 (RT2). Mucogingival surgery included the preparation of the recipient site with a tunneling protocol, where apical muscular attachment was left undisturbed to isolate the flap from the movement of the tongue during normal function. As the goal was to not coronally advance the tunneled flap, the interdental papillae were not elevated and left intact, further optimizing blood supply. A free gingival graft was harvested, de-epithelialized extra-orally, and the resulting connective tissue graft (CTG) was fed through the tunnel and stabilized with sling sutures. Partial root coverage was achieved ranging from 50% to 90% at 4 months, consistent with the initial diagnosis of RT2. There was also a visually appreciable increase in gingival thickness and in the vestibular depth.

Conclusion

A de-epithelialized CTG via tunneling without disturbing the deeper muscular attachment is a conservative method to improve phenotype of lingual recession defects.

Key points

Why is this case new information?
  • There are only a few case reports that describe soft tissue augmentation of lingual recession defects, of which none describe a tunneling technique without coronal advancement of the flap to treat a long span of multiple recession defects. This case report introduces a minimally invasive technique to increase the functional resistance of mandibular anterior lingual recession defects to plaque and calculus.
What are the keys to successful management of this case?
  • Control of gingival inflammation before and after surgery, with regular maintenance visits and oral hygiene instructions.
  • Precise tunneling, leaving deeper muscular attachment on the floor of the mouth undisturbed.
  • Connective tissue graft of even thickness that is fibrous in quality.
What are the primary limitations to success in this case?
  • A shallow lingual vestibule will not allow the clinician to leave deeper muscular attachment apical to tunneling undisturbed.
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通过微创隧道移植去表皮结缔组织,改善舌侧退缩缺损的牙龈表型:病例报告。
背景:本病例报告描述了一种微创技术,用于增强下颌前牙舌侧退缩缺损对炎症的功能性抵抗力。目前仅有少数病例报告描述了舌侧牙龈退缩的软组织增量术,其中没有一个病例报告描述了无需冠状推进皮瓣的隧道技术来治疗长跨度的多发退缩缺损。由于邻近舌头、龈缘、重要结构、原有的薄表型以及手术中的有限通道,对舌侧退缩缺损进行软组织增量具有挑战性:一名 30 岁的男性被转诊治疗 22-27 号牙齿舌面的牙龈退缩,诊断为 2 型牙龈退缩(RT2)。黏膜龈手术包括用隧道方案准备受术部位,不破坏根尖肌肉附着物,以将皮瓣与正常功能时舌头的运动隔离开来。由于目标是不在冠状面上推进隧道瓣,因此没有抬高牙间乳头,而是保持完整,从而进一步优化血液供应。采集游离牙龈移植物,进行口外去表皮处理,然后将所得结缔组织移植物(CTG)送入隧道,并用吊带缝合固定。4 个月后,部分牙根覆盖率达到 50%-90%,与 RT2 的初步诊断一致。牙龈厚度和前庭深度也有明显增加:结论:在不破坏深层肌肉附着的情况下,通过隧道法去除上皮的 CTG 是改善舌侧退缩缺损表型的保守方法:为什么本病例是新信息?目前仅有少数病例报告描述了舌侧退缩缺损的软组织增量术,其中没有一个病例报告描述了在不对皮瓣进行冠状推进的情况下采用隧道技术治疗长跨度的多发退缩缺损。本病例报告介绍了一种微创技术,用于增加下颌前牙舌侧退缩缺损对牙菌斑和牙结石的功能阻力。本病例成功治疗的关键是什么?术前和术后控制牙龈炎症,定期复诊并指导口腔卫生。精确打隧道,不破坏口底深层的肌肉附着。厚度均匀的纤维结缔组织移植。该病例成功的主要限制因素是什么?舌前庭较浅,临床医生无法不破坏隧道顶端的深层肌肉附着。
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来源期刊
Clinical Advances in Periodontics
Clinical Advances in Periodontics DENTISTRY, ORAL SURGERY & MEDICINE-
CiteScore
1.60
自引率
0.00%
发文量
40
期刊最新文献
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