Sakthivel Sankaran, M. Rajasekaran, V. Govindaraj, P. Sowmiya, S. ShinyRebekka, B. Kaleeswaran
{"title":"Acute Cyanide Poisoning: Identification of Prussic Acid in by Analyzing of Various Parameters in Cattle","authors":"Sakthivel Sankaran, M. Rajasekaran, V. Govindaraj, P. Sowmiya, S. ShinyRebekka, B. Kaleeswaran","doi":"10.1109/ICCSP48568.2020.9182090","DOIUrl":null,"url":null,"abstract":"Cyanide is one of the most vigorous toxins which contains in plants naturally and it can affect all the animals especially in cattle. Cogitative can be easily affected by this toxic substance from consuming various kinds of natural plants like sorghum and Johnson grass. Most of the plant species contains cyanogenic glycosides and these cyanogenic glycosides are converted to hydrogen cyanide by hydrolyzation in the cattle body. After the hydrolyzation, hydrogen cyanide combine with methemoglobin is hemoglobin in the form of metalloprotein and this composite constrain the oxidative phosphorylation. As a ramification of this process the affected cogitative animals may die. This toxic cyanide can affect animals rapidly. In olden process the cyanide detection can be identified using the cherryred color change of the blood of affected cattle and in the necropsy early formation of death attendance are the cardinal symptoms of cyanide poisoning. If the treatment performed rapidly, toxin can be neutralized but in the most cases the animals die due to rapidly acting nature of the toxin. It is more important to prevent cattle from the toxic substances.","PeriodicalId":321133,"journal":{"name":"2020 International Conference on Communication and Signal Processing (ICCSP)","volume":"26 1","pages":"0"},"PeriodicalIF":0.0000,"publicationDate":"2020-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"1","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"2020 International Conference on Communication and Signal Processing (ICCSP)","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.1109/ICCSP48568.2020.9182090","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 1
Abstract
Cyanide is one of the most vigorous toxins which contains in plants naturally and it can affect all the animals especially in cattle. Cogitative can be easily affected by this toxic substance from consuming various kinds of natural plants like sorghum and Johnson grass. Most of the plant species contains cyanogenic glycosides and these cyanogenic glycosides are converted to hydrogen cyanide by hydrolyzation in the cattle body. After the hydrolyzation, hydrogen cyanide combine with methemoglobin is hemoglobin in the form of metalloprotein and this composite constrain the oxidative phosphorylation. As a ramification of this process the affected cogitative animals may die. This toxic cyanide can affect animals rapidly. In olden process the cyanide detection can be identified using the cherryred color change of the blood of affected cattle and in the necropsy early formation of death attendance are the cardinal symptoms of cyanide poisoning. If the treatment performed rapidly, toxin can be neutralized but in the most cases the animals die due to rapidly acting nature of the toxin. It is more important to prevent cattle from the toxic substances.