Evidence of Stable Isotope 13C Causing All Cancers

Reginald B. Little
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Abstract

Evidence for a general mechanism of genesis of all cancers is given by presenting a puzzle and using recent results of aspartame and acesulfame-K to fit the pieces of the cancer puzzle together for proof of the mechanism of oncogenesis. Aspartame and acesulfame-K have been linked to cancer. Aspartame is a modified dipeptide (methylated phenylalanine (modified) and aspartic acid), so modified amino acids were thought to be more innocuous. The author previously noted isotopically enriched and/or clumped bond-specific amino acids and oligonucleotides have different biochemical reactions for different biology. Acesulfame-K is not a peptide or oligonucleotide but it has some similar chemical structure to oligonucleoside and reversibly decomposes to affect oligonucleosides and affect cells and bacteria cells like E Coli for producing isotopically enriched amino acids indirectly. E. Coli is present in the human digestive system and is known to accelerate stable isotope enrichment of amino acids with 13C, 15N, and/or 17O. Thereby acesulfame-K and aspartame induce isotopic enrichments and excessive 13C, 15N, and/or 17O introductions into proteins and nucleic acids. Such effects of 13C isotopes as induced by the bacteria and these chemicals like aspartame and acesulfame-K may be coupled to effects of static magnetic field(s) and radio frequency electromagnetic waves for explaining the complexity of results and inconsistent results from separating these effects. Thereby the 13C isotope enriched phenylalanine and/or aspartic acid may be the basis of malfunctioning normal cells for the genesis of cancer. Further support for this theory of 13C causing cancer by ingestion of aspartame is given by also noting the mystery and confounding results of monosodium glutamate causing cancer as the glutamate is synthesized by bacterial culture and the resulting ingested glutamic acid enriched with 13C and 15N and possibly 17O causes adverse biochemistry and enzymatics (relative to glutamate from healthy plants and animals rather than the bacterial industrial glutamate in MSG) for cancer and other diseases as explaining the observation of cancer and other diseases from acute and chronic MSG ingestion over months and years. The third case of bacterial production of insulin during the last 25 years for recent correlation to insulin causing cancer relative to less cancer from older methods of insulin production is more evidence of bacterial production of amino acids and peptides causing enriched heavy isotopes and the resulting enriched amino acids and peptides causing cancer relative to ingestion of 13C enriched carbohydrates.
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稳定同位素13C导致所有癌症的证据
所有癌症发生的一般机制的证据是通过提出一个难题,并利用阿斯巴甜和安赛胺- k的最新结果将癌症难题的各个部分拼凑在一起,以证明肿瘤发生的机制。阿斯巴甜和安赛蜜k与癌症有关。阿斯巴甜是一种修饰的二肽(甲基化苯丙氨酸(修饰)和天冬氨酸),因此修饰的氨基酸被认为是更无害的。作者先前指出,同位素富集和/或聚集的键特异性氨基酸和寡核苷酸对不同的生物学有不同的生化反应。乙酰氨基磺酸k不是肽或寡核苷酸,但它与寡核苷具有一些相似的化学结构,并可可逆分解影响寡核苷,间接影响细胞和细菌细胞如大肠杆菌产生同位素富集氨基酸。大肠杆菌存在于人体消化系统中,已知它能加速含有13C、15N和/或17O的氨基酸的稳定同位素富集。因此,乙酰磺胺- k和阿斯巴甜诱导同位素富集和过量的13C、15N和/或17O引入蛋白质和核酸。细菌和阿斯巴甜和安赛胺- k等化学物质诱导的13C同位素的这种影响可能与静态磁场和射频电磁波的影响相耦合,以解释分离这些影响的结果的复杂性和不一致的结果。因此,13C同位素富集的苯丙氨酸和/或天冬氨酸可能是癌症发生的正常细胞功能失调的基础。进一步支持13C通过摄入阿斯巴甜致癌的理论,还注意到谷氨酸钠致癌的神秘和令人困惑的结果,因为谷氨酸是通过细菌培养合成的,由此摄入的富含13C和15N以及可能的17O的谷氨酸会导致不良的生物化学和酶作用(相对于来自健康植物和动物的谷氨酸,而不是味精中的细菌工业谷氨酸),从而导致癌症和其他疾病解释了癌症和其他疾病在几个月和几年的时间里由急性和慢性味精摄入引起的观察。在过去的25年里,细菌生产胰岛素的第三个案例与胰岛素致癌的相关性相对于旧的胰岛素生产方法较少的癌症是更多的证据,表明细菌生产氨基酸和肽导致富集的重同位素以及由此产生的富集的氨基酸和肽导致癌症相对于摄入富含13C的碳水化合物。
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