Retraction: “MicroRNA-205 affects mouse granulosa cell apoptosis and estradiol synthesis by targeting CREB1”

IF 3 3区生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY Journal of cellular biochemistry Pub Date : 2024-04-03 DOI:10.1002/jcb.30553

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Abstract

Retraction: “MicroRNA-205 affects mouse granulosa cell apoptosis and estradiol synthesis by targeting CREB1,” by Pengju Zhang, Jun Wang, Hongyan Lang, Weixia Wang, Xiaohui Liu, Haiyan Liu, Chengcheng Tan, Xintao Li, Yumin Zhao, Xinghong Wu, J Cell Biochem. 2018; 120: 8466-8474: The above article, published online on 16 December 2018 in Wiley Online Library (https://onlinelibrary.wiley.com/doi/full/10.1002/jcb.28133) has been retracted by agreement between the journal's Editor in Chief, Christian Behl, and Wiley Periodicals LLC.

The retraction has been agreed following an investigation based on allegations raised by third parties. Several flaws and inconsistencies between results presented and experimental methods described were found. Furthermore, multiple image duplications as well as image elements that were published previously in a different scientific context were found in Figures 2, 3, and 5. Thus, the editors consider the conclusions of this article to be invalid. The authors did not respond when asked to collaborate during the investigation and confirm the retraction.

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撤回："MicroRNA-205通过靶向CREB1影响小鼠颗粒细胞凋亡和雌二醇合成

撤稿："MicroRNA-205 通过靶向 CREB1 影响小鼠颗粒细胞凋亡和雌二醇合成》，作者：张鹏举、王军、郎红艳、王伟霞、刘晓辉、刘海燕、谭成成、李新涛、赵玉敏、吴星红，《细胞生化杂志》。2018; 120: 8466-8474:经期刊主编 Christian Behl 和 Wiley Periodicals LLC 协议，2018 年 12 月 16 日在线发表于 Wiley Online Library (https://onlinelibrary.wiley.com/doi/full/10.1002/jcb.28133) 的上述文章已被撤回。根据第三方提出的指控进行调查后，双方同意撤回文章。在对第三方提出的指控进行调查后，双方同意撤回论文。调查发现，论文所提供的结果与所描述的实验方法之间存在若干缺陷和不一致之处。此外，在图 2、图 3 和图 5 中还发现了多处图像重复以及以前在不同科学背景下发表过的图像元素。因此，编辑认为这篇文章的结论无效。当被要求在调查期间进行合作并确认撤稿时，作者没有做出回应。

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来源期刊

Journal of cellular biochemistry 生物-生化与分子生物学

CiteScore

9.90

自引率

0.00%

发文量

164

审稿时长

1 months

期刊介绍： The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.