{"title":"Corrigendum to “PDGFB as a vascular normalization agent in an ovarian cancer model treated with a gamma-secretase inhibitor”","authors":"","doi":"10.1002/jcp.31354","DOIUrl":null,"url":null,"abstract":"<p>Maria C. Pazos, Gonzalo Sequeira, Sebastian Bocchicchio, Maria May, Dalhia Abramovich, Fernanda Parborell, Marta Tesone, Griselda Irusta. J Cell Physiol 2018 Aug;233(8):5949-5961. 10.1002/jcp.26404. Epub 2018 Feb 27</p><p>In the original version of this article, the authors mistakenly used the wrong image to display the “control” panel in Figure 2b, resulting in a duplication with the “DAPT” panel depicting cleaved caspase-3 staining in Figure 3. The correct Figure 2b is presented below.</p><p>Additionally, the legend for Figure 1 is corrected to (changes in bold):</p><p>PDGFB treated tumor growth and inmunohistochemical analysis of ovarian xenograft tumors. (a) SKOV3 cells were inoculated in female nude mice. When tumors were palpable the animals received rec-PDGFB as treatment or saline solution (control) as described in Material and Methods section. Tumor size was measured in both groups. The variable was analyzed using Student's <i>t</i> test. No statistical difference was found between groups. (b–e) show hematoxylin and eosin stained sections of each tumor belonging to all experimental groups (control, rec-PDGFB, DAPT, and DAPT+rec-PDGFB). Scale bars: 20 μm in ×10 magnification pictures and 50 μm in ×40 magnification insets. <b>Note that insets do not always correspond to the same histological section shown at ×10 magnification.</b></p><p>This correction doesn't change the results and conclusions. The authors apologize for any confusion these errors may have caused.</p>","PeriodicalId":15220,"journal":{"name":"Journal of Cellular Physiology","volume":"239 11","pages":""},"PeriodicalIF":4.0000,"publicationDate":"2024-07-18","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1002/jcp.31354","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of Cellular Physiology","FirstCategoryId":"99","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1002/jcp.31354","RegionNum":2,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q2","JCRName":"CELL BIOLOGY","Score":null,"Total":0}
引用次数: 0
Abstract
Maria C. Pazos, Gonzalo Sequeira, Sebastian Bocchicchio, Maria May, Dalhia Abramovich, Fernanda Parborell, Marta Tesone, Griselda Irusta. J Cell Physiol 2018 Aug;233(8):5949-5961. 10.1002/jcp.26404. Epub 2018 Feb 27
In the original version of this article, the authors mistakenly used the wrong image to display the “control” panel in Figure 2b, resulting in a duplication with the “DAPT” panel depicting cleaved caspase-3 staining in Figure 3. The correct Figure 2b is presented below.
Additionally, the legend for Figure 1 is corrected to (changes in bold):
PDGFB treated tumor growth and inmunohistochemical analysis of ovarian xenograft tumors. (a) SKOV3 cells were inoculated in female nude mice. When tumors were palpable the animals received rec-PDGFB as treatment or saline solution (control) as described in Material and Methods section. Tumor size was measured in both groups. The variable was analyzed using Student's t test. No statistical difference was found between groups. (b–e) show hematoxylin and eosin stained sections of each tumor belonging to all experimental groups (control, rec-PDGFB, DAPT, and DAPT+rec-PDGFB). Scale bars: 20 μm in ×10 magnification pictures and 50 μm in ×40 magnification insets. Note that insets do not always correspond to the same histological section shown at ×10 magnification.
This correction doesn't change the results and conclusions. The authors apologize for any confusion these errors may have caused.
期刊介绍:
The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.