RETRACTION: "Coinhibition of S1PR1 and GP130 by siRNA-loaded Alginate-conjugated Trimethyl Chitosan Nanoparticles Robustly Blocks Development of Cancer Cells".

IF 4.5 2区 生物学 Q2 CELL BIOLOGY Journal of Cellular Physiology Pub Date : 2024-11-01 Epub Date: 2024-07-22 DOI:10.1002/jcp.31376
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引用次数: 0

Abstract

Retraction: N. Rostami, A. Nikkhoo, Y. Khazaei-poul, S. Farhadi, M. Sadat Haeri, S. Moghadaszadeh Ardebili, N. Aghaei Vanda, F. Atyabi, A. Namdar, M. Baghaei, N. Haghnavaz, T. Kazemi, M. Yousefi, G. Ghalamfarsa, G. Sabz, F. Jadidi-Niaragh, "Coinhibition of S1PR1 and GP130 by siRNA-loaded Alginate-conjugated Trimethyl Chitosan Nanoparticles Robustly Blocks Development of Cancer Cells," Journal of Cellular Physiology 235, no. 12 (2020): 9702-9717, https://doi.org/10.1002/jcp.29781. The above article, published online on 18 May 2020 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Alexander Hutchison; and Wiley Periodicals LLC. The retraction has been agreed due to concerns raised by third parties on the data presented in the article. Specifically, the spectra in Figure 2c and 2d display irregularities suggesting data manipulation or fabrication. The data provided by the corresponding author upon request was inadequate to address the concerns. Therefore, the editors consider the conclusions of this article to be invalid. The authors have been informed of the decision of retraction.

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转载:《siRNA负载的藻酸盐结合三甲基壳聚糖纳米颗粒对S1PR1和GP130的联合抑制可有效阻断癌细胞的发育》。
Retraction:N. Rostami, A. Nikkhoo, Y. Khazaei-poul, S. Farhadi, M. Sadat Haeri, S. Moghadaszadeh Ardebili, N. Aghaei Vanda, F. Atyabi, A. Namdar, M. Baghaei, N. Haghnavaz, T. Kazemi, M. Yousefi, G. Ghalamfarsa, G. Sabz, F. Jadidi-Niaragh.Jadidi-Niaragh, "Coinhibition of S1PR1 and GP130 by siRNA-loaded Alginate-conjugated Trimethyl Chitosan Nanoparticles Robustly Blocks Development of Cancer Cells," Journal of Cellular Physiology 235, no..上述文章于 2020 年 5 月 18 日在线发表于 Wiley Online Library (wileyonlinelibrary.com),经期刊主编 Alexander Hutchison 和 Wiley Periodicals LLC 协议,该文章已被撤回。同意撤稿的原因是第三方对文章中提供的数据提出了疑虑。具体来说,图 2c 和 2d 中的光谱显示不规则,表明数据被篡改或捏造。通讯作者应要求提供的数据不足以解决这些问题。因此,编辑认为这篇文章的结论无效。撤稿决定已通知作者。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
CiteScore
14.70
自引率
0.00%
发文量
256
审稿时长
1 months
期刊介绍: The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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